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Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

Early treatment with the right dose of stem cells can reduce lupus symptoms.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Patients with RASopathies are at risk for autoimmune disorders and should be routinely screened.

Folliculotropic mycosis fungoides has unique molecular features and cell interactions that could guide targeted therapy.

Patients with RASopathies have a higher risk of autoimmune disorders and should be routinely screened.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

Different immune responses cause hair loss in scalp diseases, with unique patterns in scalp psoriasis possibly protecting against hair loss.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

Certain types of T cells are essential for healthy skin and play a role in skin diseases, but more research is needed to improve treatments.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

Activating Notch in adult skin causes T cells and neural crest cells to gather, leading to skin issues.

Diffuse alopecia areata involves more inflammation and higher allergy-related antibodies than patchy types.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

People with alopecia areata have more Th17 cells and fewer Treg cells, which may be key to the condition's development.

Treg dysfunction is linked to various autoimmune skin diseases, and understanding Treg properties is key for new treatments.

Mesenchymal stromal cells may help treat severe COVID-19, but more research is needed to confirm their effectiveness.

HSP90 and lamin A/C are crucial for hair growth and could be targets for treating hair loss.

Early regulatory T cells are crucial for normal skin pigmentation.

The study suggests that a specific type of immune cell, memory-like NK cells, may increase during active hair loss in Alopecia areata.

ILC1 cells contribute to hair loss in alopecia areata.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

Understanding how hair follicle stem cells work can help find new ways to prevent hair loss and promote hair growth.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

Different immune cells like platelets, mast cells, neutrophils, macrophages, T cells, B cells, and innate lymphoid cells all play roles in skin wound healing, but more research is needed due to inconsistent results and the complex nature of the immune response.