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Tiny particles from stem cells help activate hair growth cells and encourage hair growth in mice without being toxic.

Applying a special compound can promote hair growth without harmful side effects.

KGF and activin are crucial for skin healing and repair.

Too much Smad7 can cause serious changes in skin tissues, including problems with hair growth, thymus shrinkage, and eye development issues.

Melatonin helps regulate hair growth and protects the hair follicle from stress.

Green tea component EGCG could potentially promote human hair growth.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

The TGM3 gene's promoter region is key for skin and hair cell function and may aid gene therapy.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

FGF signaling is a promising target for developing treatments for wounds, metabolic diseases, and cancer.

Pure carbon nanotubes are safe for mice, but impure ones cause immune issues and hair loss.

Laminin-511 is crucial for early hair growth and maintaining important hair development signals.

Fullerenes show potential in skin care but need more safety research.

Scientists turned mouse stem cells into skin cells that can grow into skin layers and structures.

Cystatin M/E strongly inhibits cathepsin V and cathepsin L, important for skin formation.

The study found that two enzymes linked to hair loss are located in different parts of the scalp, supporting a common treatment's effectiveness.

Scientists created stem cells that can grow hair and skin.

Thymosin β4 promotes hair growth by activating stem cells in hair follicles.

Finasteride effectively blocks rat enzymes, but with varying methods and strength.

Different soft tissue fillers can cause various skin reactions; biodegradable fillers are safer and non-biodegradable ones like silicone can lead to long-term problems.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Grafted rodent and human cells can regenerate hair follicles, but efficiency decreases with age.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Androgens block hair growth by disrupting cell signals; targeting GSK-3 may help treat hair loss.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

Low-oxygen conditions and ECM degradation products increase the healing abilities of perivascular stem cells.

Prolactin affects hair growth cycles and can cause early hair follicle regression.

The Wnt/β-catenin pathway is important for tissue development and has potential in regenerative medicine, but requires more research for therapeutic use.

New compounds called benzoquinolinones may treat conditions linked to excess DHT.