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Researchers developed a mouse model that tracks hair growth using bioluminescence, improving accuracy in studying hair cycles.

Wnt1/βcatenin signaling is crucial for heart repair after injury.

Wnt signaling controls whether hair follicle stem cells stay inactive or regenerate hair.

The telogen phase of hair growth is active and important for preparing hair follicles for regeneration, not just a resting stage.

Chemokine signaling is important for hair development.

Hormones during pregnancy and lactation keep skin stem cells inactive, preventing hair growth.

STAT5 activation is crucial for starting the hair growth phase.

The environment around melanocyte stem cells is key for hair regeneration and color, with certain injuries affecting hair color and potential treatments for pigmentation disorders.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

Stem cell self-renewal is complex and needs more research for full understanding.

Understanding how melanocyte stem cells work could lead to new treatments for hair graying and skin pigmentation disorders.

Macrophages help hair growth after injury through CX3CR1 and TGF-β1.

Melanocytes are diverse cells important for pigmentation and skin health, influenced by genetics and environment.

NF-κB signaling is crucial in many diseases and can be targeted for new treatments.

New therapies show promise for wound healing, but more research is needed for safe, affordable options.

WNT10B is linked to cancer development and affects survival and disease progression in various cancers.

The Hairless gene is crucial for healthy skin and hair growth.

Hair follicle stem cells in hairpoor mice are disrupted, causing hair loss.

A certain signaling pathway in mice, when increased, causes hair to gray by depleting the cells that give hair its color.

The document concluded that stem cells are crucial for skin repair, regeneration, and may help in developing advanced skin substitutes.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

The conclusion suggests that focusing on certain cellular pathways may improve the prevention and repair of hair loss caused by radiotherapy.

PDGF signaling is crucial for maintaining fat stem cells in the skin, and its level of activation can either preserve these cells or cause fibrosis.

Glycyrrhizic acid may help reduce unwanted hair growth.

Tectoridin helps human hair cells grow and makes mouse hair longer, suggesting it could treat hair loss.

Different body areas in mice produce different hair types due to interactions between skin layers.

Special particles from skin cells can promote hair growth by activating a specific growth signal.

Bmpr2 and Acvr2a receptors are crucial for hair retention and color.

UV exposure causes hair thinning, graying, and changes in hair growth cycles in mice.

Melatonin affects gene expression in goat hair follicles, potentially increasing cashmere production.