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Scarring alopecia affects different hair follicle stem cells than nonscarring alopecia, and the infundibular region could be a new treatment target.

Hair follicle stem cells could help repair nerves and avoid ethical issues linked to embryonic stem cells.

Mice with human skin protein K8 had more skin problems and cancer.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Advancements in creating skin grafts with biomaterials and stem cells are promising, but more research is needed for clinical application.

Low-dose UVB light improves hair growth effects of certain stem cells by increasing reactive oxygen species.

The gelatin/β-TCP scaffold with nanoparticles improves wound healing and skin regeneration.

Changing Wnt signaling can lead to more or less hair growth and might help treat hair loss and skin conditions.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Fetal wounds heal without scarring because of different biological factors, which could help improve adult wound healing.

The conclusion is that future hair loss treatments should target the root causes of hair thinning, not just promote hair growth.

The 3D scaffold helped maintain hair cell traits and could improve hair loss treatments.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Cell-based therapies show promise for treating Limbal Stem Cell Deficiency but need more research.

SCF and ET-1 together significantly increase skin pigmentation and melanin production.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

Macrophage-stimulating protein helps hair grow and can start hair growth phase in mice and human hair samples.

DHT stops hair regrowth in mice, similar to human hair loss.

FoxN1 overexpression in young mice harms immune cell and skin development.

Scientists have made progress in understanding hair follicle stem cells, identifying specific genes and markers, and suggesting their use in treating hair and skin conditions.

Melanoma development can be linked to the breakdown of skin's melanin-producing units.

Sebaceous glands in our skin, developing during pregnancy and active in puberty, produce sebum for skin lubrication, temperature control, and fighting germs, also help in hormone regulation, and their dysfunction can cause conditions like acne and hair loss.

Chrysanthemum zawadskii extract helps hair grow by stimulating hair cells.

Scaffold improves hair growth potential.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Topical treatments can deliver active molecules to skin stem cells, potentially helping treat skin and hair disorders, including skin cancers and hair loss.

Fluocinolone acetonide slows down hair follicle stem cells but speeds up skin cell growth in mice.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

The mesenchyme can start hair growth, but the exact signal that causes this is still unknown.