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miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

Researchers found genes that control hair color and growth change before the visible coat color changes in snowshoe hares.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

Finasteride-treated male rats' offspring had altered glucose metabolism, potentially increasing diabetes risk.

The most different genetic segment between Africans and East Asians is the EDA2R/AR region, with two main types influenced by population changes and natural selection, and linked to baldness.

Eating a high-glycemic diet may worsen acne by increasing certain protein levels and expressions in the skin.

ELL is crucial for gene transcription related to skin cell growth.

A boy's growth and immune problems were caused by a new mutation in the STAT5B gene.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Estrogen Receptor ß (ERß) is the main hormone controller in human skin and hair follicles, not Estrogen Receptor α (ERα) or the Androgen Receptor (AR).

Certain genetic variations in the AR and ERβ genes can affect androgen levels in women.

Androgens may influence bladder cancer progression by affecting cellular behavior.

Reduced AR gene methylation may cause early pubic hair growth in girls.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

Desmoglein 4 is controlled by specific proteins that affect hair growth.

Phosphorylation controls TFEB's location in the cell, affecting cell metabolism and stress response.

Melatonin improves cashmere goat hair quality by increasing follicles and reducing skin aging.

A gene called BMAL1 plays a role in controlling hair growth.

The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

Hirsute women have lower type 2 17β-HSD enzyme levels, which improve with treatment.

The conclusion is that androgenetic alopecia and senescent alopecia have unique gene changes, suggesting different causes and potential treatments for these hair loss types.

Mutations in the androgen receptor gene cause Androgen Insensitivity Syndrome, affecting sexual development.

Longer CAG repeats in gene linked to more severe hair loss in females.

Researchers created immortal human skin cells with constant testosterone receptor activity to study hair loss and test treatments.

Tamoxifen may cause female hair loss by increasing androgen receptor expression.

No significant link between CAG repeat numbers and female pattern hair loss in Han Chinese population.

Different substances that activate or block the androgen receptor can affect male development and treat conditions like prostate cancer.

AGA causes hair loss by shrinking hair follicles due to DHT binding, and can be treated with finasteride and minoxidil.

Male and female bodies respond differently to stress, influenced by hormones and development stages, with implications for stress-related diseases.

Androgens may cause hair loss by increasing TGF-beta1 from scalp cells, which inhibits hair cell growth.