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Certain genetic variations in the AR and ERβ genes can affect androgen levels in women.

Androgenetic alopecia is mainly caused by genetic factors and increased androgen activity, leading to hair follicle miniaturization.

Androgen receptors are active in many tissues of both male and female mice, not just reproductive organs.

Personalized treatments for hair loss are becoming more effective by using genetic information.

The study suggests that motor neurons created from stem cells of patients with spinal and bulbar muscular atrophy show signs of the disease, including changes in protein levels and cell functions.

Hirsute women have lower type 2 17β-HSD enzyme levels, which improve with treatment.

AR/EDA2R gene linked to early-onset female hair loss, but 20p11 gene not involved.

Scientists restored fertility in male mice lacking a key fertility gene by using a modified gene.

The research identified genes and non-coding RNAs in cells that could be affected by testosterone, which may help understand hair loss and prostate cancer.

DHT affects bone growth by altering gene activity in osteoblasts, potentially complicating steroid use.

Hormones and genes affect hair growth and male baldness.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Mice with human gene experienced hair loss when treated with DHT.

ERG increases SOX9, promoting prostate cancer growth and invasion.

The most different genetic segment between Africans and East Asians is the EDA2R/AR region, with two main types influenced by population changes and natural selection, and linked to baldness.

Women with greater androgen sensitivity respond better to finasteride for hair loss.

E211 G>A gene linked to lower risk of severe prostate cancer and hair loss.

Testing for CAG repeat polymorphism in the androgen receptor gene is not currently recommended for managing hypogonadism.

Shorter CAG repeats in a specific gene may increase male hormone activity and symptoms like acne and excess hair in women with PCOS.

The study found that androgen receptors in skin cells mainly affect the focal adhesion pathway and control the caveolin-1 gene, with implications for new treatments for related diseases.

Male children's genital development issues can be caused by genetic mutations or environmental factors affecting hormone action.

Five new mutations in the androgen receptor gene were found, helping to understand androgen insensitivity syndrome better.

Insufficient androgen action in male fetuses can cause genital development issues due to genetic mutations or environmental chemicals.

Mutations in certain receptors can cause diseases and offer new treatment options.

New treatments for prostate cancer are less toxic and show promise, but more research is needed to enhance their effectiveness and reduce side effects.

Effective treatments for spinal and bulbar muscular atrophy are not yet available; more research is needed.

AGA causes hair loss by shrinking hair follicles due to DHT binding, and can be treated with finasteride and minoxidil.

Higher androgen levels and site-specific AR expression cause sex-related skeletal differences, and certain steroids can boost AR expression and androgen effects in bone cells.

A genetic variant in the androgen receptor gene increases heart disease risk in women but not in men.

Androgenetic alopecia in men is genetic and linked to health issues like obesity and heart disease, with treatments including minoxidil, finasteride, and hair transplants.