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The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Hair growth phase and certain genes can speed up wound healing, while an inflammatory mediator can slow down new hair growth after a wound. Understanding these factors can improve tissue regeneration during wound healing.

Hirsute women have lower type 2 17β-HSD enzyme levels, which improve with treatment.

PGD2 increases androgen receptor activity in hair cells, which could be targeted to treat hair loss.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Male and female bodies respond differently to stress, influenced by hormones and development stages, with implications for stress-related diseases.

Hormones and their receptors, especially androgens, play a key role in hair growth and disorders like baldness.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

NcoA4 may have roles beyond helping control gene activity, possibly affecting cell behavior and stability.

Finasteride may improve prostate cancer treatment outcomes.

PRP injections safely increase hair density and thickness in androgenetic alopecia.

Androgens like testosterone affect skin health and can lead to conditions such as acne and hair loss, with various treatments available.

Genomic approach finds new possible treatments for hair loss.

Androgens and androgen receptors are important for metabolic health, affecting how the body uses glucose and fats through mitochondrial function.

MicroRNA-302 helps improve the conversion of body cells into stem cells by blocking NR2F2.

MicroRNAs are crucial for controlling skin development and healing by regulating genes.

Dihydrotestosterone causes p27 protein breakdown in ovarian and breast cancer cells by binding with SKP2.

The conclusion is that using drugs to block the TOR pathway might slow aging and prevent age-related diseases.

Male genital development is driven by androgen signaling and understanding it could help address congenital anomalies.

Effective treatments for spinal and bulbar muscular atrophy are not yet available; more research is needed.

TR3 is mainly found in hair follicle stem cells and may be involved in hair loss.

Keratins help protect cells, aid in cancer diagnosis, and influence cancer behavior and treatment.

Hedgehog signaling helps keep hair follicle stem cells the same in both young and old human skin.

Foxi3 expression in developing teeth and hair is controlled by the ectodysplasin pathway.

Poplar seed hairs grow from the placenta at the ovary base, with endoreduplication playing a key role in their development, and share similar cellulose synthesis processes with cotton fibers.

Eating less can improve stem cell function and increase lifespan.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

Dihydrotestosterone treatment on 2D and 3D-cultured skin cells slows down hair growth by affecting certain genes and could be a potential target for hair loss treatment.

Androgens are important for female fertility and could help in IVF treatment, but also play a role in causing PCOS.

Impaired androgen production in 46,XY DSDs causes ambiguous genitalia and requires long-term care.