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Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Caffeine can damage hearing cells and affect hearing recovery after ear trauma.

Mice genetically modified to produce more Del1 protein had faster hair regrowth.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Hair follicle melanocytes die during hair regression.

ΔNp63α stops TAp73β from working in skin cancer by blocking its access to specific genes, not by directly interacting with it.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

Stem cell-derived conditioned medium shows promise for treating various medical conditions but requires standardized production and further validation.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

Certain mice without specific receptors or mast cells don't lose hair from stress.

A specific enzyme is essential for proper hair follicle stem cell development and healthy skin.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

Proteins like aPKC and PDGF-AA, substances like adenosine and ATP, and adipose-derived stem cells all play important roles in hair growth and health, and could potentially be used to treat hair loss and skin conditions.

Changing how mesenchymal stromal cells are grown can improve their healing abilities.

Cyclosporine A may help treat hair loss by blocking a protein that inhibits hair growth.

Exosomes from umbilical cord cells fix hearing loss and damaged ear hair cells in mice.

PDGF signaling is crucial for maintaining and renewing hair follicle stem cells, which could help treat hair loss.

The review found that different stem cell types in the skin are crucial for repair and could help treat skin diseases and cancer.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.

Low-Level Laser Therapy can stimulate healing and cell function, potentially leading to wider medical use.

Akt2 protein is essential for normal cell division in early mouse embryos.

Synthetic ceramides may help hair growth by boosting cell growth in hair follicles.

A protein called ectodysplasin-A2 increases a hair growth inhibitor in balding cells, which could be a target for hair loss treatment.

Nuclear shape and chromatin changes affect gene expression in skin cell differentiation.

Activating TLR9 helps heal wounds and regrow hair by using specific immune cells.

Tiny particles called extracellular vesicles could help with skin healing and hair growth, but more research is needed.

β-catenin in the dermal papilla is crucial for normal hair growth and repair.

The conclusion is that understanding how feathers and hairs pattern can help in developing hair regeneration treatments.

The conclusion is that androgenetic alopecia and senescent alopecia have unique gene changes, suggesting different causes and potential treatments for these hair loss types.