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A newly found RNA in Cashmere goats may play a role in hair growth and development.

GLI2 increases follistatin production in human skin cells.

Genes and epigenetic changes are important in the development of Polycystic Ovary Syndrome.

New hair regrowth model introduced, imiquimod kills skin cancer cells, T-cadherin loss makes skin cancer more invasive, no strong link between PTCH1 gene and skin cancer after transplant, and male teens more likely to have hereditary hair loss.

New hair regrowth model proposed, imiquimod found to kill skin cancer cells, T-cadherin loss linked to invasive skin cancer, no clear gene link to skin cancer after transplant, and study on children's hair loss shows male dominance and genetic ties.

Certain genetic markers linked to reproductive potential were identified by their impact on a protein's ability to bind to genes.

Scientists restored fertility in male mice lacking a key fertility gene by using a modified gene.

BMP4 gene is crucial for hair follicle development in Liaoning cashmere goats.

Involucrin gene expression is controlled by specific proteins and signaling pathways.

The update highlights that non-classic congenital adrenal hyperplasia is common in women with excess male hormones, requires specific hormone tests for diagnosis, and has various treatment options depending on age and symptoms.

Two specific hair keratin genes are active during hair growth and decline as hair transitions to rest.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

Early diagnosis and individualized treatment improve outcomes for Congenital Adrenal Hyperplasia.

The JAK/STAT pathway is important in cell processes and disease, and JAK inhibitors are promising for treating related conditions.

Researchers found a genetic region that influences the number of coat layers in dogs.

The NF-κB effector p65/RelA activates hair keratin genes, aiding hair formation.

The VEGF +405G allele may increase the risk of PCOS in South Indian women.

Hairlessness in mammals is due to complex genetic changes in both genes and regulatory regions.

Hairless mammals have genetic changes in both their protein-coding and regulatory sequences related to hair.

The KRTAP11-1 gene promoter is crucial for specific expression in sheep wool cortex.

Hairlessness in mammals is caused by combined changes in genes and regulatory regions.

The hypothesis suggests that PCOS may start early in life due to genetic and environmental factors, influencing future reproductive and metabolic problems.

Wnt signaling controls whether hair follicle stem cells stay inactive or regenerate hair.

The AUTS2 gene is linked to neurological disorders and may affect human brain development and cognition.

Researchers found two new genetic variants linked to Alzheimer's disease.

Both gene and non-gene areas of DNA evolved to make some mammals hairless.

Foxn1 gene regulation is crucial for thymus development but not for hair growth.

Two mutations in KRT74 and EDAR genes cause sheep to have finer wool.

Mutations in specific llama genes may affect fiber quality for textiles.

VDR regulation varies by tissue and is crucial for its biological functions.