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Vitamin D is important for bones, hair, blood pressure, and breast development.

Vitamin D might be involved in the development of alopecia areata and could help in its treatment.

The document concludes that inflammation markers can be used in diabetes, vitamin D3 affects immune pathways, hyperthyroidism changes hormone levels, androgen levels help diagnose Adrenocortical Carcinoma, erectile dysfunction is linked to diabetes, hypogonadism is common in HIV-infected males, and hormones can be biomarkers for various conditions.

Vitamin D and estrogen may help protect heart and kidney health, and maintaining sufficient vitamin D levels could be especially beneficial for African Americans, postmenopausal women, and people with chronic kidney disease.

Human skin acts like a hormone-producing organ, making and managing various hormones important for skin and hair health.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Melatonin receptors in hair follicles help regulate hair growth and could treat hair loss.

Men and women respond differently to drugs for COVID-19, high cholesterol, and diabetes, which suggests a need for personalized treatments.

People with Alopecia Areata often have lower vitamin D levels, and vitamin D supplements might help treat it.

Some growth factors, while important for normal body functions, can cause diseases when not regulated properly.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

The method can measure vitamin B3 levels in human hair accurately.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

DPP4-positive fibroblasts play a major role in producing proteins that lead to skin fibrosis.

Removing REDD1 in mice increases skin fat by making fat cells larger and more numerous.

Further research is needed to understand how the microbiome affects hair loss in Alopecia Areata.

Fractional photothermolysis helps wounds heal with minimal scarring.

BRG1 is essential for skin cells to move and heal wounds properly.

Adequate vitamin D is crucial for skin health and should be considered in dermatology.

Too much zinc in the diet can cause hair loss and color change in young mice by reducing copper in the body.

Hair follicle culture helps develop new treatments for hair loss.

Women with early-onset baldness should be checked for early bone loss, but bone density doesn't change with baldness severity.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

OCT binds strongly to hair sheath cells and may affect skin and hair growth with fewer side effects than vitamin D3.

Vitamin D receptor is crucial for bone health and mineral metabolism.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.