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Vitamin D deficiency is common in people with certain types of hair loss, like alopecia areata and female pattern hair loss.

VDR regulation varies by tissue and is crucial for its biological functions.

Higher leptin and lower vitamin D levels may contribute to male pattern baldness.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

An imbalance between certain immune cells is linked to a chronic skin condition and may be influenced by obesity, smoking, and autoimmune issues.

The vitamin D receptor can work without its usual activating molecule.

Small amounts of Vitamin D3 can boost hair growth, but too much can stop it.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

Adding a specific gene to skin cells can help treat skin disorders like psoriasis.

Long-term finasteride use doesn't change bone density or metabolism.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Vitamin D and its receptor may help prevent skin cancer.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

A girl had rickets due to a gene mutation affecting vitamin D response.

Vitamin D is crucial for bone health and preventing serious diseases.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.

Hair loss can be a key sign of a rare type of rickets when vitamin D treatment doesn't work and advanced tests aren't available.

Vitamin D receptor is crucial for skin wound healing.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Oral Vitamin D can improve hair density and reduce hair loss in Telogen Effluvium patients.

Researchers created a mouse model of a type of rickets that does not cause hair loss.