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The conclusion is that hair growth can be improved by activating hair cycles, changing the surrounding environment, healing wounds to create new hair follicles, and using stem cell technology.

Modifying certain signals in the body can help wounds heal without scars and regrow hair.

FGF9 from certain T cells helps create new hair follicles during wound healing, which could potentially be used for hair loss treatments.

Mice without the IL-6 gene had more hair growth after injury due to higher activity of a related protein, Stat3.

Wounds can regenerate hair in young mice, but this ability declines with age, offering insights for improving tissue regeneration in the elderly.

FGF9 from certain cells can trigger new hair growth during wound healing, but humans have fewer of these cells, which may limit hair regrowth.

A substance called FGF9 from certain immune cells can trigger new hair growth during wound healing in mice, but humans may not have the same response due to fewer of these cells.

Careful selection of mice by genetics and age, and controlled housing conditions improve the reliability of hair regrowth in wound healing tests.

Prostaglandin D2 blocks new hair growth after skin injury through the Gpr44 receptor.

After skin injury, adult mice can grow new hair follicles, and this process can be increased or stopped by manipulating Wnt signals.

Wounding may stimulate hair growth, but more research is needed to confirm the safety and effectiveness of related treatments.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

Bacteria can help skin regenerate through a process called IL-1β signaling.

Wound healing can help prevent hair loss from chemotherapy in young rats by increasing interleukin-1β signaling.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

M2 macrophages, a type of immune cell, help in new hair growth on scars by producing growth factors.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

An 80-year-old patient grew new hair on a wound, showing that elderly people can still regenerate hair.

Activating the Sonic hedgehog pathway can help regenerate hair follicles during wound healing in mice, potentially improving regeneration after injury.

Different cell types work together to repair skin, and targeting them may improve healing and reduce scarring.

Thymic stromal lymphopoietin (TSLP) promotes hair growth, especially after skin injury.

Mice without collagen VI have slower hair growth normally but faster regrowth after injury.

β-Catenin is essential for new hair growth after skin injury.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

Blocking a protein called CXXC5 with a specific peptide can stimulate hair regrowth and new hair growth in wounds.

Boosting HGF signaling could improve the creation of hair follicles in lab-made skin.

Full thickness wounds on Lanyu pigs' skin resulted in abnormal skin structure and function due to changes in molecular expression patterns.

Artificial dermal template treatment can stimulate complete skin and hair follicle regrowth.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

Targeting CXXC5 and GSK-3β may help treat male pattern baldness.