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The conclusion is that tissue structure is key for stem cell communication and maintaining healthy tissues.

Skin has specialized touch receptors that can tell different sensations apart.

Certain mutations in the H6PD gene cause Cortisone Reductase Deficiency by affecting hormone production.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Activin helps skin growth and healing mainly through stromal cells and affects keratinocytes based on its amount.

White lupin uses specific genes to grow root hairs and access phosphorus when it's scarce.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

Muscles and nerves that cause goosebumps also help control hair growth.

SHISA6 helps maintain certain stem cells in mouse testes by blocking signals that would otherwise cause them to differentiate.

Some common medications can harm male fertility, but many effects can be reversed.

Fat cells are important for healthy skin, hair growth, and healing, and changes in these cells can affect skin conditions and aging.

Mutations in Gasdermin A3 cause skin inflammation and hair loss by disrupting mitochondria.

A20 protein is crucial for normal skin and hair development.

After menopause, women lose a lot of skin collagen, but estrogen replacement might improve skin health.

Women with PCOS often have skin problems like excessive hair, acne, hair loss, and dark patches, which can be treated with hormonal and non-hormonal therapies.

Targeting the skin's endocannabinoid system could help treat skin disorders.

The circadian clock influences the behavior and regeneration of stem cells in the body.

Cells in hair follicles help create fat cells in the skin by releasing a protein called Sonic Hedgehog.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Sphynx cats are hairless and Devon Rex cats have curly hair due to specific genetic mutations.

The document concludes that early recognition and treatment of PCOS in adolescents is crucial for managing symptoms and long-term health risks.

The conclusion is that hair growth can be improved by activating hair cycles, changing the surrounding environment, healing wounds to create new hair follicles, and using stem cell technology.

The HR protein's role as a repressor is essential for controlling hair growth.

Mutations in the DSG4 gene can cause a rare hair disorder similar to monilethrix.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Wnt signaling is crucial for skin and hair development and its disruption can cause skin tumors.

Progress has been made in skin and nerve regeneration, but more research is needed to improve methods and ensure safety.

Women with PCOS have higher androgen levels that decrease with age but are still higher than in women without PCOS.

mTOR signaling helps activate hair stem cells by balancing out the suppression caused by BMP during hair growth.

Women with PCOS have different levels of certain fats and proteins in their blood, which could help diagnose the condition.