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French maritime pine bark extract significantly increased hair density in menopausal women.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

Deleting part of a gene in mice causes wavy hair and high pup loss.

Early recognition and management of skin side effects from new cancer therapies can prevent treatment delays.

Rapamycin treatment helps with mitochondrial disease by reducing PKC levels.

Stem cells help remove dead cells to keep tissues healthy by balancing cell replacement and clearance.

Blocking JAK3 signaling can reverse hair loss from alopecia areata.

The genes KRT25 and SP6 affect curly hair in horses, with KRT25 also causing hair loss. If both genes are mutated, the horse gets curly hair and hair loss. KRT25 can hide the effect of SP6.

Cats lose fur due to various reasons, including allergies, infections, genetics, hormones, diet, cancer, stress, and some conditions are treatable while others are not.

A gene mutation in Lama3 is linked to a common type of hair loss.

People with severe hair loss (Alopecia Areata) have different scalp bacteria than healthy people, which might help predict the condition's progress.

Activating Nrf2 in skin cells causes skin disease similar to chloracne in mice.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

The TRPV3 gene variant may cause the long-haired suri alpaca coat.

PPARγ is essential for maintaining healthy skin, controlling inflammation, and ensuring proper skin barrier function.

Wnt, Eda, and Shh pathways are crucial for different stages of sweat gland development in mice.

New chemotherapy drugs cause skin side effects, but treatments like minocycline and tetracycline can help reduce them.

A new non-invasive method can analyze skin mRNA to understand skin diseases better.

Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

The document concludes that non-endocrine alopecia in pets varies in treatment effectiveness and often has a poor prognosis, especially in cats.

Removing a methyl group from the ITGAV gene speeds up bone formation in a specific type of bone disease model.

Future research should focus on making bioengineered skin that completely restores all skin functions.

Animal research has greatly advanced dermatology.

Combining systemic and topical treatments, guided by weekly fungal cultures, effectively treats cat ringworm.

Gene therapy has potential as a future treatment for Hutchinson-Gilford progeria syndrome.

Proper control of β-catenin activity is crucial for development and preventing diseases like cancer.

Wound covers with α-13'-COOH from vitamin E can improve and speed up wound healing.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Autophagy prevents early aging and maintains lipid and pheromone balance in mouse glands.

Fibroblast state switching is crucial for skin healing and development.