Search

everythinglearnresearchcommunity

for

Search:↓

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Removing Dicer from pigment cells in newborn mice causes early hair graying and changes in cell migration molecules.

DNA methylation likely doesn't cause different lambskin patterns in Hu sheep.

Adult mouse skin contains stem cells that can create new hair, skin, and oil glands.

The gene Foxn1 is important for hair growth, and understanding it may lead to new alopecia treatments.

Mice lacking a key DNA methylation enzyme in skin cells have a lower chance of activating stem cells necessary for hair growth, leading to progressive hair loss.

Skin can heal wounds without hair follicle stem cells, but it takes a bit longer.

CaBP1 and CaBP2 are necessary for proper hearing and neurotransmission in the ear's inner hair cells.

Keeping β-catenin levels high in mammary cells disrupts their development and branching.

Hair, teeth, and mammary glands develop similarly at first but use different genes later.

Skin patterns are formed by simple reaction-diffusion mechanisms.

TNF family proteins are crucial for the development of skin features like hair, teeth, and mammary glands.

Low-level laser therapy combined with Neoptide improved hair regrowth better than either treatment alone in rats.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Mouse hair follicle cells briefly grow during the early hair growth phase, showing that these cells are important for starting the hair cycle.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

Future research on ectodysplasin should explore its role in diseases, stem cells, and evolution, and continue developing treatments for genetic disorders like hypohidrotic ectodermal dysplasia.

CaBP1 and 2 are important for maintaining the activity of calcium channels necessary for hearing in inner ear cells.

CaBP1 and CaBP2 are necessary for proper hearing and neurotransmission in the ear's inner hair cells.

Noggin is necessary to start the hair growth phase in skin after birth.

Using CRISPR for gene editing in the body is promising but needs better delivery methods to be more efficient and specific.

Pannexin 3 helps skin and hair growth by controlling a protein called Epiprofin.

Early development of hair, teeth, and glands involves specific signaling pathways and cellular interactions.

Foxi3 expression in developing teeth and hair is controlled by the ectodysplasin pathway.

The skin's basement membrane is specially designed to support different types of connections between skin layers and hair follicles.

Mice studies show that Protein Phosphatase 2A is crucial for cell growth, development, and disease prevention.

Scientists found a specific group of itch-sensing nerve cells in mice important for feeling itch but not for sensing heat or touch.

Chrysanthemum zawadskii extract may be a better treatment for hair loss than Minoxidil.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

A gene deletion causes the "hairless" trait in Iffa Credo rats.