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Balanced vitamin D levels are crucial to delay aging and prevent related diseases.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Low Vitamin D is common in people with Alopecia Areata, but its impact on the condition needs more research.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

Women with frontal fibrosing alopecia have similar vitamin D levels to those with other hair loss types, suggesting vitamin D is not a key factor in this condition.

Vitamin D is essential for bone health and may reduce risks of several diseases.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Hairless protein can block vitamin D activation in skin cells.

People with alopecia areata often have lower levels of vitamin D, zinc, and folate, but more research is needed to understand if supplements can help treat it.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

The vitamin D receptor can work without its usual activating molecule.

Kids with early graying hair often have low levels of calcium, ferritin, and vitamin D3.

Vitamin D and calcium are essential for normal hair growth.

High Nesfatin-1 and low Vitamin D may increase blood pressure and heart rate in women with PCOS.

Researchers found that certain lipids, especially vitamin D3, are lower in prematurely grey hair than in pigmented hair.

Higher vitamin D levels are linked to higher iron levels in Korean women without metabolic syndrome, but not in those with it.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

The skin has multiple layers and cells, serves as a protective barrier, helps regulate temperature, enables sensation, affects appearance, and is involved in vitamin D synthesis.

Premature graying of hair is linked to vitamin D and B12 deficiencies, thyroid issues, iron imbalance, stress, and lifestyle habits, and affects quality of life.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Low levels of vitamin B12, ferritin, and calcium are linked to premature graying of hair.

Women with early-onset baldness should be checked for early bone loss, but bone density doesn't change with baldness severity.

Hormones significantly affect skin health, with vitamin D playing a key role.

Alexander the Great's engineers built a causeway using a natural sand bridge, some tiger moths avoid bats by mimicking toxic relatives' sounds, early metabolism and RNA enzymes support the ancient RNA world theory, and vitamin D is crucial for hair growth and its absence can cause baldness.