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Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A girl with rickets and hair loss was found to have a rare condition where her body didn't respond to vitamin D treatment.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

Vitamin D receptor is crucial for starting hair growth after birth.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Early diagnosis and treatment of hereditary vitamin D-resistant rickets (HVDRR) are crucial to prevent growth issues and other health problems.

Certain gene variations in the Vitamin D receptor may increase the risk of chronic hair loss.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

Two sisters with rickets and hair loss had a genetic issue with vitamin D processing, and only improved when given phosphorus supplements.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Removing part of the vitamin D receptor stops vitamin D from working properly.

A genetic defect in vitamin D receptors causes severe rickets and hair loss in children, but some heal as they age.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Vitamin D is important for bones, hair, blood pressure, and breast development.

CCCA is a common scarring hair loss in African descent women, possibly linked to genetics, hair care practices, and health issues like diabetes.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Low vitamin D might be linked to certain types of hair loss, and supplements could help, but more research is needed.

People with alopecia areata had lower vitamin D levels, but these levels didn't relate to many aspects of the condition.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D and calcium are important for quick and effective skin wound healing.

Oral vitamin D treatment can help some children with alopecia areata who have vitamin D deficiency.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Vitamin D is linked to hair health, but more research is needed for its use in treating female hair loss.

Vitamin D receptor helps control hair growth genes in skin cells.

Low iron levels are a significant risk factor for hair loss, while high vitamin D levels might be a response to hair loss, not a cause.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.