1,25-Dihydroxyvitamin D Resistance, Rickets, and Alopecia

    November 1984 in “ The American Journal of Medicine
    Ze’ev Hochberg, A Benderli, Joseph Levy … David Feldman
    Studysummary This study observed that children with a genetically transmitted defect in the 1,25-dihydroxyvitamin D3 receptor experienced spontaneous healing of rickets as they aged, despite persistent mineral imbalances during treatment.
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    Research cited in this study 4

    1. Rickets and Alopecia with Resistance to 1,25-Dihydroxyvitamin D: Two Different Clinical Courses with Two Different Cellular Defects ˜The œJournal of clinical endocrinology and metabolism/Journal of clinical endocrinology & metabolism · 1983
    2. Vitamin D Resistant Rickets With Alopecia: A Form of End Organ Resistance to 1,25 Dihydroxy Vitamin D Clinical endocrinology · 1981
    3. An Unusual Form of Vitamin D-Dependent Rickets in a Child: Alopecia and Marked End-Organ Hyposensitivity to Biologically Active Vitamin D The Journal of Clinical Endocrinology & Metabolism · 1980
    4. Rickets With Alopecia: An Inborn Error of Vitamin D Metabolism ˜The œjournal of pediatrics/˜The œJournal of pediatrics · 1979