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The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

The review found no clear link between vitamin D receptor gene variations and polycystic ovary syndrome.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Vitamin D receptor helps control hair growth genes in skin cells.

Vitamin D deficiency is common in women with PCOS and linked to some metabolic problems, but not the main cause of their metabolic issues.

Many people in Majmaah city, Saudi Arabia, don't know much about vitamin D deficiency and don't practice good vitamin D habits, with few linking it to hair loss.

People with alopecia areata had lower vitamin D levels, but these levels didn't relate to many aspects of the condition.

The study found no link between vitamin D deficiency and psoriasis but suggests more research is needed.

Vitamin D is crucial for health, and deficiency can cause hair loss and other health issues.

Most women with depression in the study had low vitamin D levels.

A girl's hair-pulling condition improved greatly after she started vitamin D treatment.

Low vitamin D and ferritin levels are linked to female hair loss.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Black patients with a specific type of hair loss have a much higher chance of lacking enough vitamin D.

Vitamin D deficiency is common in people with certain types of hair loss, like alopecia areata and female pattern hair loss.

Vitamin D Receptor is crucial for normal skin and hair growth.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

A vitamin D receptor mutation causes rickets and affects immune responses.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Hairless protein can block vitamin D activation in skin cells.

Vitamin D receptor may help protect against UV-induced skin cancer.

The vitamin D receptor can work without its usual activating molecule.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.