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Mice lacking Insig proteins had hair growth problems due to cholesterol buildup, but this was fixed by the drug simvastatin.

The protein CCN2 controls hair growth by affecting hair follicle formation and stem cell activity in mice.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Dihydrotestosterone (DHT) stops hair growth in mice by lowering a growth factor important for hair.

Mice can regenerate ear tissue without the p53 protein.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

Using radiation to make mice's hair turn gray helps study and find ways to prevent or reverse hair graying.

Blocking the FGF5 gene in sheep leads to more fine wool and active hair follicles due to changes in certain cell signaling pathways.

The document concludes that assessing hair follicle damage due to cyclophosphamide in mice involves analyzing structural changes and suggests a scoring system for standardized evaluation.

Raspberry ketone may help grow hair and improve skin elasticity.

Decorin helps hair cells grow and move, and keeps hair growth phase going in mice.

Only skin melanocytes, not other types, can color hair in mice.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Mice hair growth patterns get more complex with age and can change with events like pregnancy or injury.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

Gene therapy in mice increased lifespan and improved health without causing cancer.

Mice treatments didn't grow hair, a patient treatment may affect immune response, and people with hair loss often feel anxious or depressed.

Stress can stop hair growth in mice, and treatments can reverse this effect.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Mice with more Flightless I protein grew back their claws better after amputation.

Beta-caryophyllene helps improve wound healing in mice, especially in females.

New hair can grow from large wounds in mice, but less so as they age, involving reprogramming of skin cells and specific molecular pathways.

The Itpr3 gene causes a specific hair pattern in mice.