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Rumex japonicus Houtt. may be an effective treatment for atopic dermatitis by reducing inflammation.

Platelet-Rich Plasma and stem cell therapy can increase hair count and density, but the best method for preparation and treatment still needs to be determined.

Miscanthus sinensis flower extract may help promote hair growth and prevent hair loss.

TNC+ fibroblasts play a key role in skin inflammation by interacting with T cells.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

Researchers found four key stages of cell development that are important for hair growth and shedding in cashmere goats.

Older hair follicle stem cells have a reduced ability to renew themselves, leading to more hair loss.

Activating Nrf2 helps wounds heal faster by increasing hair follicle stem cells.

People with alopecia areata have more Th17 cells and fewer Treg cells, which may be key to the condition's development.

New methods to improve the healing abilities of mesenchymal stem cells for disease treatment are promising but need more research.

Icariin helps mouse hair grow by boosting a growth factor in skin cells.

The role of γδT-cells in causing alopecia areata remains unclear.

SH-MSCs gel reduced IL-6 and increased TGF-β, suggesting it could treat alopecia.

SH-MSCs gel can effectively treat alopecia by increasing IL-10 and decreasing TNF-α gene expression.

NKG2D+CD4+ T cells are higher in alopecia areata patients and may be involved in the disease.

The document concludes that there is a significant lack of reporting on the sex and age of cells in skin research, which could affect clinical trials and treatments.

ILC1 cells contribute to hair loss in alopecia areata.

The meeting presented new findings on hair stem cells, pigmentation, genetics, and modern hair treatment techniques.

The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

Tph cells are linked to the severity of systemic lupus erythematosus.

Air pollution (PM10) increases skin inflammation and aging by reducing collagen and may trigger a repair response in skin cells.

An imbalance between certain immune cells is linked to a chronic skin condition and may be influenced by obesity, smoking, and autoimmune issues.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

Understanding how hair follicle stem cells work can help find new ways to prevent hair loss and promote hair growth.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Some patients with mycosis fungoides or Sézary syndrome experience hair loss, which may be similar to alopecia areata or linked to skin lesions, possibly due to abnormal T cells, and bexarotene can help treat it.

Mast cells likely promote skin scarring and fibrosis, but their exact role is still unclear.