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Eating a high-fat fish oil diet caused mice to lose hair due to a specific immune cell activity in the skin linked to a protein called E-FABP.

Shh and Dhh affect skin development and can cause tumors, while Ihh does not.

Psoriasis involves immune and genetic factors, and understanding these can improve treatments.

Blocking DGAT1 reduces oil gland size in mice and dogs, but only mice experience hair loss.

Small molecule DMF improves psoriasis and multiple sclerosis, adult skin cells can be made to grow new hair, certain skin cells initiate hair growth, IL-17C controls gut health and can cause skin inflammation, and skin cells produce IL-17 that can lead to psoriasis.

Fatty acid transport protein 4 is essential for skin and hair development.

AP-2α and AP-2β proteins are essential for healthy adult skin and hair.

Pangolins have lost some skin-related genes, but kept others, leading to their unique scales and skin features.

Glucosylceramides are essential for healthy skin and proper wound healing.

Transplanting growing hair follicles into scars can help regenerate and improve scar tissue.

Not having enough or having too much of the protein Grainyhead-like 3 leads to various developmental problems.

A specific gene variant is linked to heart disease, increased heart muscle, curly hair, and thick skin on palms and soles.

A specific immune response helps control mite populations on the skin, maintaining healthy hair follicles.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

Mice with human skin protein K8 had more skin problems and cancer.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.

Activating Notch in adult skin causes T cells and neural crest cells to gather, leading to skin issues.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

Vitamin D receptor is crucial for starting hair growth after birth.

K16 can partially replace K14 but causes hair loss and skin issues.

Natural gene therapy shows promise for treating skin disorders like epidermolysis bullosa.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Autophagy helps control skin inflammation and cancer responses and regulates hair growth by affecting stem cell activity.

Tight junctions are key for skin protection and controlling what gets absorbed or passes through the skin.

Changing light exposure can affect hair growth timing in goats, possibly due to a key gene, CSDC2.