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A substance called VIP might protect hair follicles from being attacked by the immune system, and problems with VIP signaling could lead to hair loss in alopecia areata.

The hair keratin gene KRT81 is found in both normal and breast cancer cells and helps them invade surrounding tissues.

Aquaporins could be new drug targets for treating polycystic ovary syndrome.

A new MBTPS2 gene variant disrupts fat metabolism and collagen production, causing Osteogenesis imperfecta.

Certain mutations in the KLHL24 gene cause a skin disorder by breaking down an important skin protein.

The PON1 192R gene variant is linked to a higher risk of psoriasis and heart disease in Western Mexico.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

NcoA4 may have roles beyond helping control gene activity, possibly affecting cell behavior and stability.

Removing a methyl group from the ITGAV gene speeds up bone formation in a specific type of bone disease model.

Chronic stress in mice changes skin metabolism and gene expression, leading to hair loss.

TCDD speeds up skin barrier formation by increasing certain gene expressions.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

S100A4 and S100A6 proteins may activate stem cells for hair follicle regeneration and could be potential targets for hair loss treatments.

SOX9 helps determine stem cell roles by interacting with DNA and proteins that control gene activity.

PLAU and SerpinB2 affect cell death differently in various forms of leprosy and could be targets for new treatments.

Found microRNA differences in hair cells, suggesting potential treatment targets for hair loss.

Cells from certain embryo parts can induce head formation in another embryo, involving complex signaling pathways.

Hair follicles in the back of the rosette fancy mouse have reversed orientations due to a gene mutation.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Different sPLA2 enzymes affect immunity, skin and hair health, reproduction, and may be potential targets for therapy.

The document reports findings on genetic research, including ethical concerns about genome editing, improved diagnosis of mitochondrial mutations, solving inherited eye diseases, confirming gene roles in epilepsy, linking a gene to aneurysms, and identifying genes associated with age-related macular degeneration.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.

A20 protein is crucial for normal skin and hair development.

Future research on ectodysplasin should explore its role in diseases, stem cells, and evolution, and continue developing treatments for genetic disorders like hypohidrotic ectodermal dysplasia.

EDA signaling is linked to skin disorders, various cancers, and liver disease.

Notch signaling is essential for healthy skin and hair follicle maintenance.

Smad-4 and Smad-7 are key in hair follicle development, with other Smads being less important.

Mice healed without scars as fetuses but developed scars as adults, suggesting scarless healing might be replicated with further research.

Baricitinib reduces inflammation and improves cell health in premature aging cells.