Search

everythinglearnresearchcommunity

for

Search:↓

Male hormones affect oil-producing skin cells differently based on their body location, and the drug spironolactone can reduce these effects.

Different compounds move through artificial sebum at different rates, which can help choose the best ones for targeting hair follicles.

MEK and BRAF inhibitors increase sebum production and accumulation, which could cause acne-like side effects.

The sebaceous gland has more roles than just producing sebum and contributing to acne, and new research could lead to better skin disease treatments.

Genetic changes in mice help understand skin and hair disorders, aiding treatment development for acne and hair loss.

Acne is caused by increased sebum, abnormal skin shedding, bacteria, and inflammation, not dirt; treatments vary from creams to antibiotics or isotretinoin, with severe cases needing a dermatologist's care.

Men with androgenetic alopecia have different scalp oils and microbes compared to those without.

Hormones and signaling pathways control sebaceous gland function and could help treat acne.

Engineered skin substitutes can grow hair but have limitations like missing sebaceous glands and hair not breaking through the skin naturally.

The new drug delivery system releases the drug better in sebum and targets follicles more effectively than the conventional cream.

Sebaceous glands play a key role in skin health, immunity, and various skin diseases.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

Alopecia areata causes varying hair loss patterns, affecting hair, nails, and possibly glands, with treatment outcomes depending on disease duration and extent.

A topical treatment safely and effectively reduced acne by causing targeted cell death in sebaceous glands without side effects.

VEGFR-2 is active in hair follicles, sebaceous glands, sweat glands, and skin on the human scalp.

Horse skin has a layered epidermis, a dermis with hair follicles, sweat and sebaceous glands, and is supplied by small arteries.

The study found that sweat glands normally suppress immune responses, but this is disrupted in certain skin diseases, possibly contributing to their development.

TGFβ signaling prevents sebaceous gland cells from producing fats.

DHEA stimulates skin oil glands and could help postmenopausal women, with potential for acne and excessive hair growth treatments.

Different women's hair and skin glands respond to hormones in varied ways, which can cause unwanted hair growth even with normal hormone levels, and more research is needed to treat this effectively.

Loss of sebaceous glands and inflammation may contribute to the development of scarring alopecia.

The best way to treat acne is to prevent healthy skin glands from turning into acne lesions by controlling the triggers early on.

Pregnancy can cause skin pigmentation, stretch marks, and changes in hair, nails, and sweat glands, with most resolving after birth.

The model suggests that scalp tension could lead to hair loss, with factors like blood vessel hardening, enlarged oil glands, and poor microcirculation also playing a role. It also hints at a possible link between skull shape and baldness pattern.

MPZL3 protein helps control the size of oil glands and the growth of oil-producing cells in both mice and humans.

Acne severity is linked to higher levels of certain androgenic hormones, even when those levels are within the normal range.

Isotretinoin effectively reduces acne but causes dry skin, higher skin pH, more redness, and hair loss.

UV-B light increases inflammation-related substances in acne-related skin cells.

A new non-invasive method can analyze skin mRNA to understand skin diseases better.

Oral itraconazole effectively reaches and treats deep skin layers in guinea pigs.