Search

everythinglearnresearchcommunity

for

Search:↓

The meeting highlighted important advances in stem cell research and its potential for creating new medical treatments.

CD201+ fascia progenitors are essential for wound healing and could be targeted for treating skin conditions.

Mouse zigzag hair bends form due to a 3-day cycle of changes in hair progenitors and their environment.

PNKP is essential for keeping adult mouse progenitor cells healthy and growing normally.

Adult esophageal cells can start to become like skin cells, with a key pathway influencing this change.

Hair follicles in the back of the rosette fancy mouse have reversed orientations due to a gene mutation.

Sebaceous glands can heal and regenerate after injury using their own stem cells and help from hair follicle cells.

miR-29 is a key factor that accelerates aging.

The skin's basement membrane is specially designed to support different types of connections between skin layers and hair follicles.

Stem cells help remove dead cells to keep tissues healthy by balancing cell replacement and clearance.

Hdac1 and Hdac2 help maintain and protect the cells that control hair growth.

CD4+ skin cells may be precursors to basal cell carcinoma.

DNA methylation is essential for skin and hair follicle development, and could be a target for treating skin diseases.

A new technology showed that the SOX9 gene might control heart scar formation after injury, suggesting new treatment possibilities.

The document concludes that identifying the specific cells where skin cancers begin is important for creating better prevention, detection, and treatment methods.

Skin cell types develop when specific genes are turned on by removing certain chemical tags from DNA.

Activating β-catenin in mammary cells leads to changes that cause early-stage abnormal growths similar to skin structures.

HDAC6 helps keep ovarian follicles dormant, extending female fertility.

Scientists made a mouse that shows how a specific protein in the skin changes and affects hair growth and shape.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

The mesenchyme can start hair growth, but the exact signal that causes this is still unknown.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

Hair growth-related cells need the enzyme SCD1 to help maintain the area that supports hair growth.

MicroRNA-205 helps hair grow by changing the stiffness and contraction of hair follicle cells.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

Dermal EZH2 controls skin cell growth and differentiation in mice.

Skin and hair renewal is maintained by both fast and slow cycling stem cells, with hair regrowth primarily driven by specific stem cells in the hair follicle bulge. These cells can also help heal wounds and potentially treat hair loss.