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Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Human hair follicle cells can grow hair when put into mouse skin if they stay in contact with mouse cells.

The telogen phase of hair growth is active and important for preparing hair follicles for regeneration, not just a resting stage.

Hair growth is influenced by hormones and goes through different phases; androgens can both promote and inhibit hair growth depending on the body area.

The hair follicle is a great model for research to improve hair growth treatments.

NF-κB is crucial for different stages and types of hair growth in mice.

Follistatin helps hair growth and cycling, while activin prevents it.

Vitamin D receptor is crucial for starting hair growth after birth.

Prolactin slows down hair growth in mice.

Leptin helps start the growth phase of hair.

Certain proteins and their receptors are more active during the growth phase of human hair and could be targeted to treat hair disorders.

EGF controls hair growth by regulating hair follicles' growth phases.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Hair follicles can be used to quickly assess drug effects in cancer treatment.

GRK2 is essential for healthy hair follicle function, and its absence can lead to hair loss and cysts.

Some herbs and their components might help treat hair loss by affecting various biological pathways, but more research and regulation are needed.

Bcl-2 overexpression protects against UVB damage but worsens hair loss from chemotherapy.

The chapter explains the causes of excessive hair growth and masculinization in women and how to measure hormone levels related to these conditions.

Hair follicle stem cells are promising for wound healing but require more research for safe clinical use.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

The document concludes that assessing hair follicle damage due to cyclophosphamide in mice involves analyzing structural changes and suggests a scoring system for standardized evaluation.

Healthy mitochondria in skin cells are essential for proper hair growth and skin cell interaction in mice.

A form of vitamin E promotes hair growth by activating a specific skin pathway.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Fat tissue under the skin affects hair growth and aging; reducing its inflammation may help treat hair loss.

Chemotherapy damages hair follicles, causing hair loss and other cellular changes.

Inactive hair follicle stem cells help prevent skin cancer.

Using neurohormones to control keratin can lead to new skin disease treatments.