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Researchers found genetic mutations causing hypohidrotic ectodermal dysplasia in 88% of studied patients and identified new mutations and genetic variations affecting the disease.

Not having enough or having too much of the protein Grainyhead-like 3 leads to various developmental problems.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

The study concluded that specific proteins are necessary to maintain the structure that holds epithelial cells tightly together.

A different gene near the hairless gene on chromosome 8p21 causes a rare hair loss condition in a German family.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

Different types of sun exposure damage skin cells and immune cells, with chronic exposure leading to more severe and lasting damage.

KIF18B is important for correctly positioning cell division machinery in skin cells, affecting hair follicle development.

Researchers found a new gene area linked to male-pattern baldness, which, along with another gene, significantly increases the risk of hair loss in men.

Stem cells, especially from fat tissue and Wharton's jelly, can potentially regenerate hair follicles and treat hair loss, but more research is needed to perfect the treatment.

BMI1 is essential for preventing hair greying and maintaining hair color.

The gene for slick hair in Senepol cattle is located on chromosome 20 and may involve the SRD5A2 gene.

A20 protein is crucial for normal skin and hair development.

BAF200 is essential for proper heart and coronary artery formation.

Hair follicle development is controlled by interactions between skin tissues and specific molecular signals.

Platelet-rich plasma may help hair follicle cells grow by affecting certain genes and pathways.

Tooth papilla cells can help regenerate hair follicles and grow hair.

The study identified and characterized new keratin genes linked to hair follicles and epithelial tissues.

Certain cells in the adult mouse ear come from cranial neural crest cells, but muscle and hair cells do not.

Notch signaling disruptions can cause various skin diseases.

Dicer is crucial for hair growth in mice.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

The ZIP7 gene helps control zinc levels in cells by moving zinc from the Golgi apparatus to the cytoplasm.

Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

Abnormal scalp whorls can indicate brain development issues but may also be seen in neurologically normal people.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

Growth factors are crucial for hair development and could help treat hair diseases.

New treatments for ichthyosis, like protein replacement and gene therapy, show promise and may become standard care.

SHISA6 helps maintain certain stem cells in mouse testes by blocking signals that would otherwise cause them to differentiate.

ATP-dependent chromatin-remodeling complexes are crucial for gene regulation, cell differentiation, and organ development in mammals.