Search

everythinglearnresearchcommunity

for

Search:↓

The research identified key molecules that help hair matrix and dermal papilla cells communicate and influence hair growth in cashmere goats.

Blocking DPP4 can potentially speed up hair growth and regeneration, especially after injury or in cases of hair loss.

Certain gene variations and irregular X chromosome activity may contribute to hair loss in women who can have children.

Certain genes and X chromosome patterns may significantly contribute to the development of hair loss.

Mutations in the androgen receptor gene cause different levels of androgen insensitivity, making it hard to create simple tests for the condition.

Bioengineered nanoparticles can effectively treat hair loss by targeting specific enzymes and receptors.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

Adenosine may promote hair growth by increasing FGF-7 levels in dermal papilla cells.

SARMs may be an effective treatment for a certain type of breast cancer by blocking cancer growth and spread.

Vitamin D receptor may help protect against UV-induced skin cancer.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Lower SMAD2/3 activation predicts more severe skin cancer.

Male mice are more susceptible to autism-like changes from valproic acid than female mice.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Doxycycline can effectively treat hair loss caused by EGFR inhibitors.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

Anabolic steroids boost muscle growth, SARMs increase muscle mass and bone density without side effects, and myostatin inhibitors block a protein that stops muscle growth, but each has potential risks.

Azelaic acid helps protect hair cells from UV damage and encourages hair growth by increasing certain gene expressions and proteins.

Certain gene variations may protect against skin issues and oxidative stress in women with PCOS.

Meis2 is essential for touch sensation and proper nerve connection to touch receptors in certain skin areas of mice.

The transcription factor Meis2 is essential for touch sensation and proper nerve development in touch receptors.