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Certain small molecules can promote hair growth by activating a cellular cleanup process called autophagy.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

Activating autophagy helps skin regenerate better during tissue expansion.

Eating fewer calories may slow skin aging and improve skin health through various biological changes.

Certain small molecules that trigger cell cleanup processes can promote hair growth.

A molecule called α-ketobutyrate was found to extend lifespan and improve aging-related symptoms in worms and mice by activating certain cellular pathways and may help develop anti-aging treatments for humans.

Modified HDL can better deliver drugs and genes, potentially improving treatments and reducing side effects.

High mTORC1 activity slows hair growth and causes it to lose color.

Rapamycin increased survival in mice with severe chronic graft-versus-host disease by expanding regulatory T cells.

Collagen VII helps skin heal and stay strong, sirolimus may lower skin cancer risk in kidney transplant patients, high-molecular-mass hyaluronan helps naked mole rats resist cancer, dermal γδ T cells aid in hair growth in rodents, and overexpression of IL-33 in mouse skin causes itchiness, offering a model for studying allergic inflammation treatments.

Blocking mTORC1 activity could increase hair pigmentation and potentially reverse greying.

The microneedle device with rapamycin and epigallocatechin gallate effectively promoted hair regrowth in mice.

Blocking mTORC1 may help prevent skin cancer by stopping the growth of certain skin stem cells.

The document concludes that understanding genetic mutations in the PI3K-AKT-mTOR pathway can lead to better diagnosis and treatment for certain genetic skin disorders.

Dihydrotestosterone (DHT) doesn't affect rat skin cell growth, but it does change cell cycle, protein levels, and other cell functions, potentially shortening hair growth cycle.

High energy boosts root hair growth in plants, while low energy stops it.

Using regulatory T cells and Rapamycin together improves chronic graft-versus-host disease treatment outcomes in mice.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

Eating a high-glycemic diet may worsen acne by increasing certain protein levels and expressions in the skin.

The mTOR pathway may be involved in the development of hair follicle tumors, with higher activity in malignant tumors.

Human sebaceous glands contain enzymes that affect androgen production and may influence sebum production and acne.

Acne patients have higher mTOR gene expression.

Metformin, usually used for diabetes, can also help treat hair loss from alopecia areata due to its ability to reduce inflammation and stimulate new hair growth.

Cow milk sugars increase fat production and inflammation in skin oil cells.

Targeted cancer therapies often cause skin problems that need careful management to improve patient quality of life and treatment success.

The study found that analyzing certain cell signaling pathways is not a reliable method to tell apart two types of skin tumors.

Heat stress reduced hair growth and affected related genes in rex rabbits.

Acne patients have higher levels of mTOR in their skin, which could be linked to future metabolic disease.

EGFR and mTOR inhibitors may help manage Olmsted syndrome symptoms.

The plant hormone auxin activates the TOR pathway, affecting gene expression related to growth and cell size.