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Different types of skin stem cells can change and adapt, which is important for developing new treatments.

Reviewers suggest clarifying the abstract, adding background on inflammation's effect on cancer, discussing immune cell roles, including counter studies, and correcting grammar.

The reviewers suggest clarifying the abstract, adding background on inflammation's effect on stem cells and cancer, discussing immune cells' role in tumors, and considering inflammation's potential to reduce cancer growth.

The reviewers suggest major revisions for clarity and inclusion of recent findings on inflammation's role in cancer.

The revised manuscript on how inflammation affects cancer stem cells was accepted after addressing issues with clarity and detail.

The reviewers suggest clarifying and expanding on the role of inflammation in stem cell-related cancer development.

The revised study on how inflammation affects cancer stem cells was accepted after improving clarity and structure.

The reviewers found the manuscript on inflammation's role in cancer stem cells to be relevant but suggested it needs clarity, more background information, discussion on immune cells' role in tumors, updated references, and grammar corrections.

Aging in hair follicle stem cells leads to hair graying, thinning, and loss.

Low oxygen areas help maintain and protect blood stem cells by using a simple sugar breakdown process for energy and managing their activity levels.

Hair follicles can regenerate after radiation damage but not during a specific growth phase.

Hair and skin can regenerate without bulge stem cells due to other compensating cells.

Wnt signaling helps control how brain stem cells divide and is important for brain repair after injury.

3,4,5-tri-O-caffeoylquinic acid promotes hair growth by activating the β-catenin pathway.

Hdac1 and Hdac2 help maintain and protect the cells that control hair growth.

Skin fat plays a key role in immune defense and healing beyond just storing energy.

NF-κB is crucial for different stages and types of hair growth in mice.

Deleting MED1 in skin cells causes hair loss and skin changes.

Fat cells are important for healthy skin, hair growth, and healing, and changes in these cells can affect skin conditions and aging.

Gene therapy, especially using atoh1, shows promise for creating functional sensory hair cells in the inner ear, but dosing and side effects need to be managed for clinical application.

The conclusion is that proper signaling is crucial for hair growth and development, and errors can lead to cancer or hair loss.

The document concludes that while lab results for hair growth promotion are promising, human trials are needed and better testing methods should be developed.

TrichoTech™ may help hair growth and skin repair by increasing fibroblast growth and activity.

The mTOR signaling pathway is crucial for hair health and targeting it may lead to new hair loss treatments.

Deleting the BRD4 protein in certain skin cells causes hair loss and skin inflammation.

The circadian clock plays a key role in hair growth and its disruption can affect hair regeneration.

Disrupted stem cell signals in hairpoor mice cause hair loss.

New research suggests that skin cell renewal may not require a special type of cell previously thought to be essential.

BMI1 is essential for preventing hair greying and maintaining hair color.

KLF4 is important for keeping hair follicle stem cells inactive.