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Aging causes hair to gray and thin, with the timing of graying varying by race, and factors like oxidative stress and genetics can lead to hair loss.

New targeted cancer drugs can cause skin side effects, and managing them requires patient education and timely care.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Certain drugs increase calcium levels in cancer cells by triggering internal calcium release.

The skin protects the body, regulates temperature, senses touch, and makes vitamin D.

The document shows the state of pharmaceutical patents as of 1999, focusing on treatments for various diseases.

Insulin resistance is a key factor in polycystic ovary syndrome, but genetics may also contribute.

Olive oil compounds may help prevent cancer in animals, but human results are mixed.

Wnt signaling is important for development and cell regulation but can cause diseases like cancer when not working properly.

Drug repositioning is becoming more targeted and efficient with new technologies, offering personalized treatment options and growing interest in the field.

Retinoids can prevent skin cancer in high-risk people but have side effects and require more research on dosing and effectiveness.

Key genes linked to hair growth and cancer were identified in hairless mice.

The Wnt signaling pathway is not a major factor in the development of keratoacanthoma, a type of skin tumor.

Polycystic Ovary Syndrome (PCOS) is a hormonal disorder in women that can cause infertility and other health issues, and it may be improved by treatments that increase insulin sensitivity.

No FDA-approved treatments for chemotherapy-induced hair loss existed in 2006; more research was needed.

A new carrier improves skin delivery of tofacitinib for treating inflammatory skin diseases.

Finasteride treatment in rats changed the expression of genes related to psychiatric and neurological functions, and these changes persisted after stopping the drug.

Different people show different symptoms for genetic diseases because of how sensitive their bodies are to small changes in important factors.

Dihydrotestosterone causes p27 protein breakdown in ovarian and breast cancer cells by binding with SKP2.

There is no cure for alopecia areata, but treatments like JAK inhibitors show promise.

New drugs targeting the JAK-STAT pathway show promise for treating inflammatory skin diseases.

Mice can regenerate ear tissue without the p53 protein.

The Wnt/β-catenin pathway helps tissue regeneration but can also cause fibrosis, and drugs that inhibit this pathway may aid in healing skin and heart tissues.

Consider benefits and risks of new alopecia treatments for safety.

XEDAR triggers a specific signaling pathway in cells.

Serenoa repens and N-acetyl glucosamine/milk proteins complex may help with hair growth and prevent hair loss.

New treatments for Alopecia Areata show promise but need to be more effective and affordable.

Spironolactone, a heart and liver drug, has new uses including cancer treatment, viral infection prevention, and skin condition improvement.

People with alopecia have shorter hair follicles and more c-kit, a stem cell factor receptor, which could predict how the condition progresses.

Chicken feather growth involves specific genes and shares similarities with hair development.