Search

everythinglearnresearchcommunity

for

Search:↓

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

MSC-derived exosomes can help treat COVID-19, hair loss, skin aging, and arthritis.

The conclusion is that grasping how cells determine their roles through evolution is key, with expected progress from new research models and genome editing.

Stress hormone corticosterone blocks a growth factor to slow down hair stem cell activity and hair growth.

Fat from the body can help improve hair growth and scars when used in skin treatments.

PDGF signaling is crucial for maintaining and renewing hair follicle stem cells, which could help treat hair loss.

Certain signals are important for reducing specific chemical markers on hair follicle stem cells during rest periods, which is necessary for healthy hair growth.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

Retinoids increase steroid sulfatase activity in leukemia cells through RARα/RXR and involves certain pathways like phosphoinositide 3-kinase and ERK-MAP kinase.

Removing a methyl group from the ITGAV gene speeds up bone formation in a specific type of bone disease model.

Lymphatic vessels help hair follicles regenerate by interacting with stem cells.

Hair follicles are complex, dynamic mini-organs that help us understand cell growth, death, migration, and differentiation, as well as tissue regeneration and tumor biology.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Small molecule IM boosts hair growth by changing stem cell metabolism.

The conclusion is that certain cell interactions and signals are crucial for hair growth and regeneration.

Wnt signaling is crucial for pigmented hair regeneration by controlling stem cell activation and differentiation.

Endoglin is crucial for proper hair growth cycles and stem cell activation in mice.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

The document concludes that certain chemicals can help maintain stem cell pluripotency and that understanding cell states is crucial for tissue regeneration.

Transplanted hair follicle stem cells can heal damaged rat intestines.

Exosomes could potentially enhance tissue repair and regeneration with lower rejection risk and easier production than live cell therapies.

Fibroblast growth factor receptor signaling controls cell development and repair, and its malfunction can cause disorders and cancer, but it also offers potential for targeted therapies.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Improving the environment and cell interactions is key for creating human hair in the lab.

Using fat stem cells and blood cell-rich plasma together improves healing in diabetic wounds by affecting cell signaling.

Cell aging can be both good and bad for tissue repair.

The conclusion is that skin and hair patterns are formed by a mix of cell activities, molecular signals, and environmental factors.

Hominis Placenta helps hair grow back by increasing cell growth and a specific growth factor.

New cell-based therapies may improve hair loss treatments in the future.

Removing centrosomes from skin cells leads to thinner skin and stops hair growth, but does not greatly affect skin cell differentiation.