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Recent findings suggest that genetic factors, immune system issues, and skin cell defects might contribute to the development of hidradenitis suppurativa.

Aging causes hair loss and graying due to stem cell decline and changes in cell behavior and communication.

Sweat hypersensitivity can cause severe skin issues in horses.

Deoxycholic acid is FDA-approved for reducing submental fat, but its mechanisms are not fully understood.

People with hypothyroidism and hair loss often have more hair and scalp issues than those without thyroid problems.

Editing the FGF5 gene in sheep increases fine wool growth.

The synthetic retinoid EC23 thickens skin and promotes hair growth more effectively and with a lower dose than natural retinoids.

Lhx2 is a crucial regulator of the Sonic Hedgehog signaling in early mouse retinal development.

Certain vitamins in wrong amounts, alcohol abuse, metals, and other toxins can cause serious brain and nerve damage.

The Wnt/β-catenin pathway is important for body functions and diseases, and targeting it may treat conditions like cancer, but with safety challenges.

Vitamin D is essential for bone health and may reduce risks of several diseases.

Involucrin gene expression is controlled by specific proteins and signaling pathways.

CRH in the skin acts like the body's stress response system, affecting cell behavior and immune activity.

Understanding and managing side effects of hedgehog pathway inhibitors can improve treatment for advanced basal cell carcinoma.

Different treatments for Hidradenitis suppurativa range from antibiotics and hormonal therapies to surgery, depending on severity.

Non-hormonal treatments should be used first for sexual dysfunction in postmenopausal breast cancer patients on aromatase inhibitors, with hormones as a second option.

Sumatriptan injections, high-flow oxygen, and verapamil effectively treat cluster headaches.

GPRC5D is linked to the formation of hair, nails, and certain tongue areas.

Notch signaling is essential for healthy skin and hair follicle maintenance.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

New therapies are being developed that target integrin pathways to treat various diseases.

Vitamin A affects hair loss and immune response in alopecia areata.

Testosterone is linked to cardiovascular risk factors and stroke, but its exact role is unclear.

Low androgen levels might delay prostate cancer but could lead to more aggressive, therapy-resistant cancers.

Lysophospholipids like LPA and S1P are important for hair growth, immune responses, and vascular development, and could be targeted for treating diseases.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Fibroblast changes in systemic sclerosis may help understand disease severity and treatment.

Insulin resistance and high male hormone levels are major causes of Polycystic Ovary Syndrome.

Krt15+ cells in the mouse intestine resist radiation and can start tumors.

Targeting Vδ1+T-cells may help treat alopecia areata.