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Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Hormones significantly influence hair growth, with conditions like hirsutism and patterned hair loss linked to hormone levels; more research is needed for full understanding.

Understanding how androgens and their receptors work can lead to improved treatments for skin diseases.

Pharmacophore models can predict liver toxicity and central nervous system toxicity, but they have limitations and specific requirements.

Excessive sun protection might cause frontal fibrosing alopecia by disrupting skin immune balance.

Vitamin D3 and its receptor are important for hair growth, and understanding receptors could help treat hair graying and skin cancer.

The plant hormone auxin activates the TOR pathway, affecting gene expression related to growth and cell size.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Progestogens are essential in menopausal hormone therapy to prevent uterine cancer and must be chosen carefully based on individual needs.

Understanding how androgens work is key for creating new treatments for prostate issues and hair/skin conditions.

FGF21 can help reduce skin inflammation caused by C. acnes.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

Women with hair loss have more androgen receptors in certain hair follicles.

Certain gene variations in the Vitamin D Receptor are linked to higher risk of female hair loss.

Flutamide caused damage to male rat reproductive organs and may affect fertility.

RORA plays a key role in controlling seasonal hair molting by affecting hair follicle cell activity.

Shorter CAG repeats may cause hair and skin issues, while longer ones may link to acne.

CAP1 helps Arabidopsis plants grow better under ammonium stress.

Men with Kennedy disease have less chance of hair loss.

Melatonin may inhibit melanoma growth and has potential as a cancer therapy aid, but its effects on human skin pigmentation need more research.

Benign Prostatic Hyperplasia may be caused by changes in how the body processes male hormones.

Hormones during puberty increase certain receptors in the brain, and this change is influenced by estrogen levels.

Red deer only have androgen receptors in neck hair cells for mane growth during breeding season.

Hormones during pregnancy and lactation keep skin stem cells inactive, preventing hair growth.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.

Antibody treatments show promise for hair loss but need more research.

Hair follicles produce and respond to melatonin, affecting hair growth and sensitivity to estrogen.

Androgens like testosterone are important hormones for both men and women, made differently in each sex and affecting the body by regulating genes and quick interactions with cell components.

Thyrotropin-releasing hormone may help control skin and hair growth and could aid in treating related disorders.