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Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

Alopecia areata involves persistent gene abnormalities and immune activity, even in regrown hair, suggesting a risk of relapse.

Melasma's causes include genetics, sun exposure, hormones, and oxidative stress, and understanding these can help create better treatments.

Uterine leiomyomas don't significantly change gene expression in the scalp of people with Central Centrifugal Cicatricial Alopecia.

The study found key factors in the cause of hidradenitis suppurativa, its link to other diseases, and identified existing drugs that could potentially treat it.

Dermal adipose tissue in mice can change and revert to help with skin health.

Hyaluronate fragments can help reverse skin thinning by working with the CD44 receptor.

Alopecia areata is caused by immune system attacks on hair follicles, often triggered by viral infections.

NK cells play a role in skin diseases like eczema and psoriasis.

When skin blisters, healing the wound is more important than growing hair, and certain stem cells mainly fix the blisters without helping hair growth.

Mast cells and CD8 T cells interact closely in skin diseases, affecting each other's behavior and contributing to conditions like psoriasis and eczema.

Eating a high-fat fish oil diet caused mice to lose hair due to a specific immune cell activity in the skin linked to a protein called E-FABP.

Type 2 diabetes slows down skin and hair renewal by blocking important stem cell activation in mice.

AIRE deficiency causes hair loss similar to alopecia areata in mice.

Mitochondrial dysfunction is linked to various skin conditions and could be a target for treatments.

Fire Needle Therapy may help bring back skin color in vitiligo by affecting cell growth signals.

MCPIP1 in myeloid cells is important for skin cancer development and healthy hair growth.

Most patients with autoimmune blistering diseases experienced some hair loss, which may be underreported and linked to disease severity.

Higher CD70 and CD27 gene expression in alopecia areata lesions predicts disease severity and activity.

Non-immune factors play a significant role in alopecia areata.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

Genetic research has advanced our understanding of skin diseases, but complex conditions require an integrative approach for deeper insight.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

ILC1 cells contribute to hair loss in alopecia areata.

Certain scalp patterns can indicate the severity and activity of hair loss in Turkish alopecia patients.

Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

FFA's causes may include environmental triggers and genetic factors.

Decreased IGF-1R expression may contribute to sacrococcygeal pilonidal sinus development.