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Damaging mutations in NFKB2 cause a severe and distinct form of primary immunodeficiency with early-onset and often ACTH-deficiency.

DAVID syndrome is a condition with immune system and hormone deficiencies, needing early diagnosis to avoid serious complications.

The study found that immune responses disrupt hair growth cycles, causing hair loss in alopecia areata.

Melatonin treatment increases a specific RNA in goat cells that boosts cashmere growth.

The study found key factors in the cause of hidradenitis suppurativa, its link to other diseases, and identified existing drugs that could potentially treat it.

The S250C variant in a gene may cause autoimmunity and immunodeficiency by impairing protein function.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

Curcumin may help reverse aging by targeting specific genes.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

A new mutation in the STING protein causes a range of symptoms and its severity may be affected by other genetic variations; treatment with a specific inhibitor showed improvement in one patient.

A certain smell receptor in hair follicles can affect hair growth when activated by a synthetic sandalwood scent.

Using a combination of specific cell cycle regulators is better for safely keeping hair root cells alive indefinitely compared to cancer-related methods.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

NF-κB signaling is crucial in many diseases and can be targeted for new treatments.

Newborn mouse skin cells can grow hair and this process can be recreated in adult cells to potentially help with hair loss.

NF-κB is crucial for zebrafish heart repair, affecting heart cell growth and repair processes.

TLR2 is important for hair growth and can be targeted to treat hair loss.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

NF-κB is crucial for different stages and types of hair growth in mice.

XEDAR triggers a specific signaling pathway in cells.

TLR2 helps control hair growth and regeneration, and its reduction with age or obesity can impair hair growth.

Skin abnormalities can indicate immunodeficiency due to shared origins with the immune system.

Cholesterol-related compounds can stop hair growth and cause inflammation in a type of scarring hair loss.

Electrospun scaffolds can improve healing in diabetic wounds.

Vav2 changes how hair follicle stem cells' genes work as they age, which might improve regeneration but also raise cancer risk.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

New resveratrol-related compounds show promise for treating various health issues but need more research for clinical use.

Chemokine signaling is important for hair development.

African American women have a higher risk of preterm delivery than Caucasian women, and inflammatory stimuli affect gene expression in cells related to PCOS, showing a heightened inflammatory state in women with PCOS.

A new mutation in the XEDAR gene might cause a rare skin condition called hypohidrotic ectodermal dysplasia.