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DAVID syndrome is a condition with immune system and hormone deficiencies, needing early diagnosis to avoid serious complications.

Damaging mutations in NFKB2 cause a severe and distinct form of primary immunodeficiency with early-onset and often ACTH-deficiency.

The S250C variant in a gene may cause autoimmunity and immunodeficiency by impairing protein function.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

The study found key factors in the cause of hidradenitis suppurativa, its link to other diseases, and identified existing drugs that could potentially treat it.

Melatonin treatment increases a specific RNA in goat cells that boosts cashmere growth.

Methamphetamine affects gene expression in rat whisker follicles, with key genes linked to addiction.

Curcumin may help reverse aging by targeting specific genes.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

A certain smell receptor in hair follicles can affect hair growth when activated by a synthetic sandalwood scent.

The study found that immune responses disrupt hair growth cycles, causing hair loss in alopecia areata.

A new mutation in the STING protein causes a range of symptoms and its severity may be affected by other genetic variations; treatment with a specific inhibitor showed improvement in one patient.

NF-κB signaling is crucial in many diseases and can be targeted for new treatments.

Using a combination of specific cell cycle regulators is better for safely keeping hair root cells alive indefinitely compared to cancer-related methods.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

NF-κB is crucial for zebrafish heart repair, affecting heart cell growth and repair processes.

TLR2 is important for hair growth and can be targeted to treat hair loss.

Newborn mouse skin cells can grow hair and this process can be recreated in adult cells to potentially help with hair loss.

XEDAR triggers a specific signaling pathway in cells.

NF-κB is crucial for different stages and types of hair growth in mice.

TLR2 helps control hair growth and regeneration, and its reduction with age or obesity can impair hair growth.

Cholesterol-related compounds can stop hair growth and cause inflammation in a type of scarring hair loss.

Electrospun scaffolds can improve healing in diabetic wounds.

Vav2 changes how hair follicle stem cells' genes work as they age, which might improve regeneration but also raise cancer risk.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

New resveratrol-related compounds show promise for treating various health issues but need more research for clinical use.

A new mutation in the XEDAR gene might cause a rare skin condition called hypohidrotic ectodermal dysplasia.

Researchers created a model to understand heart aging, highlighting key genes and pathways, and suggesting miR-208a as a potential heart attack biomarker.

Chemokine signaling is important for hair development.