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A new treatment using nanoparticles can effectively prevent and reduce hair loss caused by chemotherapy.

ILC1 cells contribute to hair loss in alopecia areata.

Dandruff is linked to increased T cells and weakened immune protection in hair follicles.

TYK2 inhibition may help treat alopecia areata by promoting hair growth and reducing immune response.

The new aptamer TAGX-0003 shows promise as an effective treatment for hair loss disorder alopecia areata.

Blocking IL-12 can help treat alopecia areata by preventing hair follicle immune issues.

Alopecia areata may be treated by restoring hair follicle immune privilege and adjusting immune responses.

Inhibiting TYK2 can restore hair growth in alopecia areata.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Alopecia areata is an autoimmune condition causing hair loss due to the immune system attacking hair follicles, often influenced by genetics and stress.

Alopecia areata is an autoimmune disease causing patchy hair loss, often with other autoimmune disorders, but its exact causes are unknown.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

Modified rat stem cells on a special scaffold improved blood vessel formation and wound healing in skin substitutes.

Somatostatin may help protect hair follicles from immune attacks.

Diphencyprone treatment increases CD8 lymphocytes in the scalp, which is associated with hair regrowth in alopecia areata patients.

Alopecia Areata is an autoimmune disease affecting hair follicles, influenced by genetic and environmental factors, with rodent models being essential for research.

Melatonin stimulates the skin components of ram's scrotum during their non-breeding season.

Interferon gamma alone can't cause alopecia areata in C3H/HeJ mice.

New protein changes may be involved in the immune attack on hair follicles in alopecia areata.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

Platelet-rich plasma treatment is not very effective for chronic severe alopecia areata.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Alopecia areata is an autoimmune condition causing hair loss, linked to genetic factors and immune system issues, with no cure yet.

The secretions of mesenchymal stem cells could be used for healing without using the cells themselves.

Calreticulin has roles in healing, immune response, and disease beyond its known functions in the endoplasmic reticulum.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

A patient with Alopecia Areata had complete hair regrowth after using the drug baricitinib.

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.

Some viruses can cause cancer by changing cell processes and avoiding the immune system; vaccines and targeted treatments help reduce these cancers.