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A genetic marker linked to a type of hair loss was found in most patients studied.

Dandruff is linked to increased T cells and weakened immune protection in hair follicles.

The document concludes that ongoing research using animal models is crucial for better understanding and treating Alopecia Areata.

Calreticulin has roles in healing, immune response, and disease beyond its known functions in the endoplasmic reticulum.

Graft-versus-host disease is a complication where donor immune cells attack the recipient's body, often affecting the skin, liver, and gastrointestinal tract.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

A woman with alopecia totalis regrew dark hair in bands after using a corticosteroid ointment.

Hair loss in mice starts with immune cells damaging hair roots before it becomes visible.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Hair follicles are valuable for regenerative medicine and wound healing.

The review highlights the importance of stem cells in hair health and suggests new treatment strategies for hair loss conditions.

A man's sudden hair loss and color change to white was diagnosed as alopecia areata and it improved on its own after six months.

Certain KIR genes in Indian SLE patients are linked to disease severity and could be biomarkers.

Alopecia areata can be reversed by JAK inhibitors, promoting hair regrowth.

Stress may contribute to hair loss in alopecia areata by affecting immune responses and cell death in hair follicles.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Skin bacteria can help wound healing by activating certain immune cells.

Innate lymphoid cells type 1 may contribute to alopecia areata.

ILC1 cells contribute to hair loss in alopecia areata.

Certain genetic markers, especially the MICA gene, are linked to alopecia areata.

Stress-related substance P may lead to hair loss and negatively affect hair growth.

Two mouse mutations cause similar hair loss despite different skin changes.

Oxidative stress plays a significant role in alopecia areata, and new treatments may include JAK inhibitors and antioxidants.

Alopecia areata may be treated by restoring hair follicle immune privilege and adjusting immune responses.

Hair follicles could help develop eye treatments by studying immune responses.

Certain HLA class II alleles increase or decrease the risk of alopecia areata.

Hepatitis B virus exposure may be linked to increased risk of alopecia areata.

UV light helped human hair transplants survive in mice without broad immunosuppression.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.