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Mice without certain skin enzymes have faster hair growth and bigger eye glands.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Modulating BMP activity changes the number, size, shape, and type of ectodermal organs.

Targeting NETs may help reduce fibrosis in Crohn's disease.

3-Deoxysappanchalcone helps human hair cells grow and stimulates hair growth in mice by affecting certain cell signaling pathways.

Loss of keratin K2 causes skin problems and inflammation.

Macrophages are vital for skin healing, hair growth, salt balance, and cancer defense.

Keratin proteins are essential for keeping the cells in the human colon healthy and stable.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

Alopecia areata is caused by immune system issues, and JAK inhibitors might help treat it.

Mice can regenerate ear tissue without the p53 protein.

Smad4 is important for healthy hair follicles because it helps produce a protein needed for hair to stick together and grow.

The glucocorticoid receptor helps protect skin from tumors and other issues.

The document concludes that pig iPSCs show promise for transplant therapies and the field is advancing in controlling cell behavior for biology and medicine.

Lipocalin-Type Prostaglandin D2 Synthase (L-PGDS) is a protein that plays many roles in the body, including sleep regulation, pain management, food intake, and protection against harmful substances. It also affects fat metabolism, glucose intolerance, cell maturation, and is involved in various diseases like diabetes, cancer, and arthritis. It can influence sex organ development and embryonic cell differentiation, and its levels can be used as a diagnostic marker for certain conditions.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A protein called IL-36γ causes skin side effects from certain cancer treatments when combined with a common skin bacteria.

Targeting ornithine decarboxylase can help prevent skin cancer.

Transamidases help form strong crosslinks in hair proteins, crucial for hair strength.

A special gene region controls the re-emergence of a primitive wool type in Merino sheep, improving their wool yield and adaptability.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Vitamin D is important for bones, hair, blood pressure, and breast development.

PTHRP agonists can stimulate hair growth, especially in damaged follicles, while antagonists may initially increase growth but ultimately inhibit it.

Certain cancer drugs interact with skin bacteria to cause acne-like rashes.

Certain genetic variations in the FST gene are linked to better wool quality in Chinese Merino sheep.

The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.

KGF and activin are crucial for skin healing and repair.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

Keeping β-catenin levels high in mammary cells disrupts their development and branching.

The document concluded that stem cells are crucial for skin repair, regeneration, and may help in developing advanced skin substitutes.