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Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

Runx1 controls fat-related genes important for normal and cancer cell growth, affecting skin and hair cell behavior.

The document concludes that understanding how hair follicles naturally die and regenerate is important for insights into organ development and could impact health and disease treatment.

TRPV4 helps cells repair tissue and reduce scarring by controlling calcium levels.

A special gene region controls the re-emergence of a primitive wool type in Merino sheep, improving their wool yield and adaptability.

The research found that blocking a gene called NEMO can potentially prevent harmful effects of aging at the cellular level.

CENPV, a new partner of CYLD, helps regulate ciliary acetylated tubulin and is overexpressed in certain skin tumors.

Fetuin A may increase collagen production and promote scarring.

The book is a detailed guide on experimental wound healing methods, useful for researchers but not for clinicians.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

Lef1 is essential for normal skin, hair growth, and healing wounds in mice.

Collagen VII helps skin heal and stay strong, sirolimus may lower skin cancer risk in kidney transplant patients, high-molecular-mass hyaluronan helps naked mole rats resist cancer, dermal γδ T cells aid in hair growth in rodents, and overexpression of IL-33 in mouse skin causes itchiness, offering a model for studying allergic inflammation treatments.

Vitamin A may influence hair loss conditions like alopecia, but more research is needed to understand how.

Keratins help protect cells, aid in cancer diagnosis, and influence cancer behavior and treatment.

Mice lacking Insig proteins had hair growth problems due to cholesterol buildup, but this was fixed by the drug simvastatin.

Parathyroid hormones are important for hair growth, but their use in treating hair loss from chemotherapy is still uncertain.

Blocking Rab27a slows hair growth, while blocking Rab27b encourages it.

A patient with untreated congenital adrenal hyperplasia had large benign tumors in her adrenal glands removed, improving her symptoms.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

Genetic mutations can cause hair growth disorders by affecting key genes and signaling pathways.

PTHrP and its receptor can control blood vessel growth and hair development in mouse skin.

Both immature and mature fat cells are important for hair growth cycles, with immature cells promoting growth and mature cells possibly inhibiting it.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

ATP increases melanin production in skin after UV exposure, with the P2X7 receptor being crucial for this process.

Local injections of nanosized rhEPO can speed up skin healing and improve quality after deep second-degree burns.

P-selectin is not the only factor that prevents scarring in fetal wound healing in mice.

CCL5 is important for the hair growth potential of human dermal papilla cells.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.