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Tofacitinib works better and is more tolerable for severe alopecia than conventional treatments and DPCP immunotherapy.

Ruxolitinib treatment may cause eyelash growth.

Several new treatments for different types of hair loss show promise in improving patient quality of life.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

Tofacitinib may be safe for COVID-19 patients with alopecia without worsening symptoms, based on two cases.

Blocking Prostaglandin D₂ may help treat hair loss.

Higher levels of certain DNAs in blood may indicate hair follicle damage in alopecia areata patients.

Blocking Prostaglandin D₂ (PGD₂) could help treat hair loss.

Gypenosides from Gynostemma pentaphyllum were found to have anti-aging effects, increasing skin collagen and reducing wrinkles.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

Fat tissue-derived cells show promise for repairing body tissues, but more research and regulation are needed for safe use.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

Alopecia areata is a common autoimmune disease affecting about 2% of people, causing significant disability and often associated with mental health issues and other autoimmune conditions.

Minoxidil is the only FDA-approved topical drug for treating male or female pattern hair loss, and other medications like finasteride and dutasteride can also increase hair growth.

Restoring hair bulb immune privilege is crucial for managing alopecia areata.

Alopecia areata treatment varies, with no optimal method established yet.

Activating the Wnt/β-catenin pathway could lead to new hair loss treatments.

Certain types of T cells are essential for healthy skin and play a role in skin diseases, but more research is needed to improve treatments.

Tofacitinib helped regrow hair in people with severe alopecia, but side effects occurred and benefits stopped after treatment ended.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Treatment with plasma rich in growth factors improved hair density and thickness for hair loss patients.

Blocking IL-12/IL-23p40 helped reverse severe hair loss in patients.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

There are still challenges in diagnosing and treating chronic skin diseases, but there is hope for future improvements.

Understanding how hair follicle stem cells work can help find new ways to prevent hair loss and promote hair growth.

Alopecia areata involves immune system imbalances that may lead to depression and anxiety.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Ginseng and its compounds may help hair growth and prevent hair loss, but more human trials are needed to confirm this.