Search

everythinglearnresearchcommunity

for

Search:↓

Frontal fibrosing alopecia shows increased inflammation and JAK-STAT pathway activity without reduced hair proteins.

Crataegi Fructus water extract helps hair grow better and thicker.

Interferon gamma alone can't cause alopecia areata in C3H/HeJ mice.

TNFα, IFNγ, and Substance P significantly affect prolactin levels in human skin, suggesting new treatments for skin and hair conditions.

Blocking interferon-gamma might help treat various autoimmune diseases.

New treatments for vitiligo may focus on protecting melanocyte stem cells from stress and targeting specific pathways involved in the condition.

Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

A teenager had both alopecia areata and vitiligo, which are rare to occur together.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

Ginsenoside Rg1 protects mouse skin from UVB damage and helps control inflammation.

ILC1 cells contribute to hair loss in alopecia areata.

New methods to improve the healing abilities of mesenchymal stem cells for disease treatment are promising but need more research.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

Astragalus membranaceus helps regrow hair and reduce hair loss symptoms.

Chronic inflammatory skin diseases are caused by disrupted interactions between skin cells and immune cells.

Higher levels of certain DNAs in blood may indicate hair follicle damage in alopecia areata patients.

Alopecia areata is an autoimmune condition causing hair loss, linked to genetic factors and immune system issues, with no cure yet.

Lithocholic acid helps hair growth and regeneration in alopecia by activating vitamin D receptors.

Trichloroethylene causes skin inflammation in mice by increasing certain immune proteins.

Blocking the protein CXCL12 with a specific antibody can increase hair growth in common hair loss conditions.

Tofacitinib regrew hair in a man with total hair loss but raised cytokine levels, needing more research on possible side effects.

NK cells play a role in skin diseases like eczema and psoriasis.

Alopecia areata severity and treatment response are linked to specific cytokine levels.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Certain immune-related proteins are higher in people with alopecia and their healthy relatives, hinting at a genetic link.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Type I interferons play a key role in the development of various skin diseases.