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Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

Alopecia areata is caused by immune system issues, and JAK inhibitors might help treat it.

Certain proteins are found at higher levels in balding areas compared to non-balding areas, suggesting a link to hair loss. This could be useful for diagnosing and treating hair loss.

Some multiple sclerosis treatments may trigger hair loss conditions like alopecia areata.

Lichen planus is a chronic autoimmune disease that is hard to treat and more common in women.

Tofacitinib regrew hair in a man with total hair loss but raised cytokine levels, needing more research on possible side effects.

IL-17 and certain immune cells are linked to more severe alopecia areata.

Booster shots of mRNA vaccines for COVID-19 increased protective antibodies without worsening autoimmune skin conditions in patients.

Blocking the CXCR3 receptor reduces T cell accumulation in the skin and prevents hair loss in mice.

Loss of OGG1 increases skin inflammation and auto-antibodies in lupus.

Anti-IL-12/IL-23 antibody therapy effectively treats psoriasiform skin lesions in IBD patients.

NK cells play a role in skin diseases like eczema and psoriasis.

Alopecia areata severity and treatment response are linked to specific cytokine levels.

Apremilast did not work for treating severe alopecia areata.

T-cells in alopecia areata scalp show abnormal regulation, leading to less inflammation.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

Old treatments for other skin conditions showed promise for hair regrowth in mice with a hair loss condition.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

NKG2D+CD4+ T cells are higher in alopecia areata patients and may be involved in the disease.

A new mouse model for vitiligo helps study immune responses and potential treatments.

Astragalus membranaceus helps regrow hair and reduce hair loss symptoms.

Chronic inflammatory skin diseases are caused by disrupted interactions between skin cells and immune cells.

ILC1 cells contribute to hair loss in alopecia areata.

Frontal fibrosing alopecia shows increased inflammation and JAK-STAT pathway activity without reduced hair proteins.

Fetal skin has unique immune cells different from adult skin.

Targeting specific T cells may help treat alopecia areata.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Higher levels of certain DNAs in blood may indicate hair follicle damage in alopecia areata patients.

Blocking TYK2 might be a new way to treat hair loss from alopecia areata.

ILC1-like cells may contribute to hair loss in alopecia areata.