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Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

The skin's layers protect, sense, and regulate the body's internal balance, but can be prone to cancer.

The document concludes that mouse models are helpful but have limitations for skin wound healing research, and suggests using larger animals and genetically modified mice for better human application.

Apoptosis is essential for human skin development and forming a functional epidermis.

The document provides a method to classify human hair growth stages using a model with human scalp on mice, aiming to standardize hair research.

Ruxolitinib effectively regrows hair in most patients with severe hair loss.

Chitosan nanofiber scaffolds improve skin healing and are promising for wound treatment.

Alopecia areata is caused by the immune system attacking hair follicles.

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.

Skin aging is due to impaired stem cell mobilization or fewer responsive stem cells.

The niche environment controls stem cell behavior and plasticity, which is important for tissue health and repair.

Stem cells in the hair follicle are regulated by their surrounding environment, which is important for hair growth.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

The skin's internal clock affects healing, cancer risk, aging, immunity, and hair growth, and disruptions can harm skin health.

Stress can cause hair loss by negatively affecting hair follicles and this effect might be reversed with specific treatments.

TRP channels in the skin are important for sensation and health, and targeting them could help treat skin disorders.

The telogen phase of hair growth is active and important for preparing hair follicles for regeneration, not just a resting stage.

Modern lifestyles, including poor diet, stress, and long-term use of certain medications, hinder the body's ability to heal from inflammation, leading to chronic diseases.

The document concludes that hair is complex, with a detailed growth cycle, structure, and clinical importance, affecting various scientific and medical fields.

Alopecia areata is caused by immune system attacks on hair follicles, often triggered by viral infections.

Skin surface lipids are important for skin health and altering them could help prevent aging and treat skin conditions.

Hair follicles help skin immune recovery after UVB exposure.

Prolactin affects hair growth and skin conditions, and could be a target for new skin disease treatments.

The document concludes that dandruff and seborrheic dermatitis are linked to inflammation and skin changes, and treating them with specific shampoos can reduce these issues.

Mammals might fail to regenerate not because they lack the right cells, but because of how cells respond to their surroundings, and changing this environment could enhance regeneration.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

Fat tissue cells are a promising option for healing various diseases, but more research is needed to ensure they are safe and effective.

Advanced human skin models improve drug development and could replace animal testing.

The document concludes that for hair and feather growth, it's better to target the environment around stem cells than the cells themselves.