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Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Hairless protein can block vitamin D activation in skin cells.

Sphynx cats are hairless and Devon Rex cats have curly hair due to specific genetic mutations.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

Hairless mammals have genetic changes in both their protein-coding and regulatory sequences related to hair.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

The document concludes that understanding the genes and pathways involved in hair growth is crucial for developing treatments for hair diseases.

Mice without the Sp6 gene have problems developing several body parts, including hair, teeth, limbs, and lungs.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Blocking a specific protein signal can make hair grow on mouse nipples.

The research suggests that a specific skin gene can be controlled by signals within and between cells and is wrongly activated in certain skin diseases.

A specific DNA region is crucial for Foxn1 gene expression in thymus cells but not in hair follicles.

Eating collagen peptides may help with skin and hair growth.

Male pattern baldness is caused by certain cells in hair follicles and could potentially be treated by targeting a specific growth factor, TGF-β1.

Hair follicles help attract immune cells to minor skin injuries.

Essential oils from Chamaecyparis obtusa may help hair grow by increasing a growth-related gene.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

Parathyroid hormones are important for hair growth, but their use in treating hair loss from chemotherapy is still uncertain.

Microneedling with 5% minoxidil improves hair loss in Chinese men by activating Wnt/ß-catenin pathway.

ILK is essential for skin development, pigmentation, and healing.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

Hair follicles help attract immune cells to the skin during stress.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.