Search

everythinglearnresearchcommunity

for

Search:↓

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

The research found that a protein called PPARg is important for the formation and healing of sebaceous glands, which can regenerate independently from hair follicles.

Cavity macrophages gather on organ surfaces but don't really invade or help repair the organs after injury.

Edar signaling is crucial for controlling hair growth and regression.

Blocking certain immune signals can reduce skin damage from radiation therapy.

Controlling Tslp can improve health in AEC syndrome patients.

Mast cells likely promote skin scarring and fibrosis, but their exact role is still unclear.

GDNF helps grow hair and heal skin wounds by acting on hair stem cells.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

Targeted cancer therapies can cause skin side effects, but activating SOS in the skin may help reduce them.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Removing certain brain receptors in mice worsens seizure severity and response to treatment during hormone withdrawal.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

BMP signaling is important for skin color, affecting melanin production, pigment spread, and cell movement.

Skin's epithelial stem cells are crucial for repair and maintenance, and understanding them could improve treatments for skin problems.

CD201+ fascia progenitors are essential for wound healing and could be targeted for treating skin conditions.

Tet1/2/3 enzymes affect hair follicle cell development by influencing BMP signaling.

Neurotrophin-4 increases calcium current in specific mouse neurons through the PI3K pathway.

Understanding the immune-related causes of Alopecia Areata has led to potential treatments like JAK inhibitors.

The research found that a specific skin cell type not only triggers hair growth but also controls hair color, and that aging can lead to hair loss and color changes.

Some wound-healing cells can turn into fat cells around new hair growth in mice.

The enzymes Tet1, Tet2, and Tet3 are important for the development of hair follicles and determining hair shape by controlling hair keratin genes.

Researchers found that the Leptin receptor is a consistent marker for hair follicle dermal cells, which may help future hair research.

Changing CDK4 levels affects the number of stem cells in mouse hair follicles.

A new tool can analyze hair to detect changes due to hormones, genetics, and aging.

Lanceolate complexes in mouse hair follicles are essential for touch and depend on specific cells for maintenance and regeneration.

Certain immune cells in the skin release a protein that stops hair growth by keeping hair stem cells inactive.

Notch signalling helps skin cells differentiate and prevents tumors.

Wounded skin cells can revert to stem cells and help heal.