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Men generally have more severe COVID-19 cases and higher death rates than women due to biological differences.

Chemotherapy damages hair follicles, causing hair loss and other cellular changes.

HOXC13 is essential for hair and nail development by regulating Foxn1.

A tripeptide-copper complex may help hair grow by increasing cell growth and decreasing cell death.

Arachidonic acid helps hair grow by increasing growth factors and improving follicle health.

The MAGE3 hypothesis for alopecia areata did not lead to a significant breakthrough.

A substance called 15-deoxy prostaglandin J2 can cause hair follicle cells to die, which might explain how prostaglandin D2 can lead to hair loss.

Skin RAGE levels are linked to inflammation and cell death.

Tetrahydroxystilbene Glucoside may help prevent hair loss by blocking certain pathways that lead to cell death.

Asiasari radix extract may be a potential treatment for melanoma because it selectively triggers cell death in melanoma cells by affecting p53 regulation.

Environmental pollutants can damage hair health and cause hair loss.

VEGFR-2 activation is likely involved in hair follicle growth, survival, and development.

A chemical called 5-Bromo-2′-deoxyuridine caused rapid hair loss in mice by killing certain skin cells through a specific cell death pathway.

Brain-Derived Neurotrophic Factor (BDNF) slows down hair growth and promotes hair follicle regression.

PRP treatment may stimulate hair growth by promoting blood vessel formation, increasing growth factors, and preventing cell death.

Activated ras can protect kidney cells from a certain substance that causes cell death.

RET mutation is important in familial medullary thyroid carcinoma, and BRAF mutation in papillary thyroid carcinoma is linked to more aggressive cancer and higher death rates.

Isotretinoin's effects and side effects, like birth defects and depression, might be due to it causing cell death in various cells.

Distinct β-catenin patterns are linked to cell growth, not cell death, in lung cancer.

Different skin diseases show unique patterns of skin cell separation, cell death, and granular layer changes.

Researchers found WNT10A to be a key gene in developing goat hair follicles.

The research found that postmortem root bands in hair are likely caused by the breakdown of a specific part of the hair's inner structure after death.

Certain sex hormones and antiandrogens can either slow down or speed up the growth of human hair follicle cells depending on their concentration.

Four genes are potential markers for hair loss condition alopecia areata, linked to a specific type of cell death.

Face-lifts can lead to complications like bleeding, nerve injury, skin death, hair loss, and rare infections, with careful surgery reducing risks.

The conditions alopecia areata, primary sclerosing cholangitis, and ulcerative colitis may be linked by shared autoimmune and cell death mechanisms.

Eczema patients have a lower skin lipid to protein ratio, older and longer-staying hospital patients are more likely to get pressure ulcers, hair loss in AGA is linked to muscle degeneration, vitamin D deficiency is common in alopecia areata and linked to its severity, standard liver tests don't effectively detect fibrosis in psoriasis patients on methotrexate, and bullous pemphigoid patients have a higher death risk but combination therapy may reduce it.

Cold atmospheric plasma may speed up wound healing and control infections.

Dying cells can help with faster healing and new hair growth by releasing a growth-promoting molecule.

Future hair loss treatments should aim to extend hair growth, reactivate resting follicles, reverse shrinkage, and possibly create new follicles, with gene therapy showing promise.