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YH0618 helps reduce chemotherapy-induced hair loss by targeting specific proteins and pathways.

Certain genes and pathways are linked to the production of finer and denser wool in Hetian sheep.

Skin tumor regression is helped by retinoic acid signaling blocking Wnt signaling.

Loss of sebaceous glands and inflammation may contribute to the development of scarring alopecia.

Burn scars form abnormally due to changes in wound healing, and more research is needed to improve treatments.

Psoriasis lesions have fewer and smaller oil glands, which might affect the condition's development.

Japanese patients with alopecia areata often have a higher BMI and consume more vitamin C, fruit, and retinol, which may affect their condition's development or severity.

The SOSTDC1 gene is crucial for determining sheep wool type.

The dermal papilla is crucial for hair growth and health, and understanding it could lead to new hair loss treatments.

sPLA2-IIA increases growth in hair follicle stem cells and cancer cells, suggesting it could be targeted for hair growth and cancer treatment.

TR3 is mainly found in hair follicle stem cells and may be involved in hair loss.

There are two types of stem cells in rodent hair follicles, each with different keratin proteins.

Intense pulsed light treatment mainly damages pigmented hair parts but spares stem cells, allowing hair to regrow.

Stem cells can create hair follicles, potentially treating permanent hair loss, and healthy skin and hair depend on mitochondrial function and special fats.

Melatonin may protect hair follicle stem cells from damage caused by chemotherapy.

Melatonin may help prevent hair loss caused by the chemotherapy drug paclitaxel.

Skin and hair renewal is maintained by both fast and slow cycling stem cells, with hair regrowth primarily driven by specific stem cells in the hair follicle bulge. These cells can also help heal wounds and potentially treat hair loss.

Wnt signaling is crucial for pigmented hair regeneration by controlling stem cell activation and differentiation.

Hair follicle stem cells can generate all hair cell types, skin, and sebaceous glands.

S100A4 and S100A6 proteins may activate stem cells for hair follicle regeneration and could be potential targets for hair loss treatments.

Blocking mTORC1 may help prevent skin cancer by stopping the growth of certain skin stem cells.

Disrupting a specific protein's function in hair follicle stem cells triggers their activation and a self-healing process.

PPARγ signaling modulation can protect hair follicle stem cells from chemotherapy-induced damage.

β-catenin is essential for stem cell activation and proliferation in hair follicles.

Scientists turned rat thymus cells into stem cells that can help repair skin and hair.

TGF-β2 helps activate hair follicle stem cells by counteracting BMP signals.

Desmoglein 3 is important for keeping hair follicle stem cells inactive and maintaining their special properties.

Certain signals are important for reducing specific chemical markers on hair follicle stem cells during rest periods, which is necessary for healthy hair growth.

Hair and skin healing involve complex cell interactions controlled by specific molecules and pathways, and hair follicle cells can help repair skin wounds.