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Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

Fat cells help recruit healing cells and build skin structure during wound healing.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

More dermal papilla cells in hair follicles lead to larger, healthier hair, while fewer cells cause hair thinning and loss.

The skin is a complex barrier for drug penetration, but understanding its structure and interactions can improve drug delivery methods.

Stem cell niches are crucial for regulating stem cell renewal and differentiation, and understanding them can help in developing regenerative therapies.

Artificial skin development has challenges, but new materials and understanding cell behavior could improve tissue repair. Also, certain growth factors and hydrogel technology show promise for advanced skin replacement therapies.

A gene called APCDD1, which controls hair growth, is found to be faulty in a type of hair loss called hereditary hypotrichosis simplex.

Certain mutations in a hair growth-related gene cause a type of genetic hair loss.

Regenerated fully functional hair follicles using stem cells, with potential for hair regrowth therapy.

Scientists made working salivary glands in mice using bioengineered cells, which could help treat dry mouth.

Blood vessels and specific genes help turn cartilage into bone when bones heal.

New research suggests that skin cell renewal may not require a special type of cell previously thought to be essential.

Different types of stem cells with unique roles exist in blood, skin, and intestines, and this variety is important for tissue repair.

Macrophages help heal nerves by aiding the maturation of Schwann cells and are important for nerve repair.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Foxp3 is crucial for regulatory T cell function, and targeting these cells may help treat immune disorders.

Fat cells in the skin help start healing and form important repair cells after injury.

Macrophages help regrow hair by activating stem cells using AKT/β-catenin and TNF.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

Skin-derived precursors in hair follicles come from different origins but function similarly.

A mother's PPARγ is crucial for preventing harmful milk that can cause inflammation and growth problems in babies.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

The document concludes that adult mammalian skin contains multiple stem cell populations with specific markers, important for understanding skin regeneration and related conditions.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

The Wnt/β-catenin pathway helps tissue regeneration but can also cause fibrosis, and drugs that inhibit this pathway may aid in healing skin and heart tissues.

Newborn mouse skin cells can grow hair and this process can be recreated in adult cells to potentially help with hair loss.

Blocking β-catenin in skin cells improves hair growth during wound healing.

NF-κB is crucial for zebrafish heart repair, affecting heart cell growth and repair processes.