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Hair follicles greatly increase caffeine absorption through the skin shortly after it's applied.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Wool follicles are complex, involving interactions between different cell types and structures.

Blocking β-catenin in skin cells improves hair growth during wound healing.

Mutant mice help researchers understand hair growth and related genetic factors.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

Hair grays due to oxidative stress and fewer functioning melanocytes.

Prostaglandin D2 blocks new hair growth after skin injury through the Gpr44 receptor.

The document concludes that for hair and feather growth, it's better to target the environment around stem cells than the cells themselves.

Hair follicle stem cells are a promising source for tissue repair and treating skin or hair diseases.

Blocking BMP signaling causes hair loss and disrupts hair growth cycles.

Certain signals and genes play a key role in hair growth and regeneration, and understanding these could lead to new treatments for skin regeneration.

Vitamin D receptor is crucial for starting hair growth after birth.

Brain hormones significantly affect hair color and could potentially be used to prevent or reverse grey hair.

Disruptions in hair follicle fibroblast dynamics can cause hair growth problems.

Brg1 is crucial for hair growth and skin repair by maintaining stem cells and promoting regeneration.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Maintaining the right amount of retinoic acid is crucial for healthy hair and skin.

Estrogens generally inhibit hair growth and improve skin quality, but their exact effects on hair follicles are complex and not fully understood.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

α3β1-integrin is crucial for maintaining normal hair follicle shape and function but not needed for the development of the surrounding skin.

Hoxc genes control hair growth through Wnt signaling.

Hair follicle regeneration possible, more research needed.

Hormones and neuroendocrine factors control hair growth and color, and more research could lead to new hair treatment options.

Changes in keratin make hair follicles stiffer.

SCF and c-Kit decrease in AGA hair follicles, possibly affecting hair pigmentation and growth.

Aging in hair follicle stem cells leads to hair graying, thinning, and loss.

The hair follicle infundibulum plays a key role in skin health and disease, and understanding it better could lead to new skin disease treatments.

Hair follicle hyperplasia is common in both benign and malignant skin lymphoproliferative disorders, with a proposed new term "pseudolymphomatous adnexitis."

Prolactin may affect hair growth differently based on gender and scalp area.